INT824

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Context Info
Confidence 0.42
First Reported 1978
Last Reported 2006
Negated 0
Speculated 0
Reported most in Abstract
Documents 3
Total Number 6
Disease Relevance 1.53
Pain Relevance 1.18

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

nucleoplasm (ESR1) enzyme binding (ESR1) DNA binding (ESR1)
protein complex (ESR1) cytoplasm (ESR1) signal transduction (ESR1)
Anatomy Link Frequency
uterine 8
joints 2
epithelial cells 2
ESR1 (Homo sapiens)
Pain Link Frequency Relevance Heat
Pain 1 100.00 Very High Very High Very High
antagonist 13 92.76 High High
agonist 4 92.12 High High
corticosteroid 1 88.36 High High
rheumatoid arthritis 2 84.48 Quite High
endometriosis 72 75.16 Quite High
pain pelvic 4 55.84 Quite High
withdrawal 4 5.00 Very Low Very Low Very Low
anesthesia 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Rheumatoid Arthritis 2 84.48 Quite High
Endometriosis (extended) 72 75.16 Quite High
Cancer 57 69.24 Quite High
Reprotox - General 3 24 56.60 Quite High
Breast Cancer 13 55.36 Quite High
Proteinuria 1 45.44 Quite Low
Polycystic Ovary Syndrome 36 5.00 Very Low Very Low Very Low
Hyperplasia 4 5.00 Very Low Very Low Very Low
Endometrial Cancer 4 5.00 Very Low Very Low Very Low
Hypertrophy 4 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
In both treatment groups a statistically significant decrease in the number of painful and/or swollen joints, an increase in haemoglobin and a decrease in ESR, serum ceruloplasmin-, alpha1-acid glycoprotein-, IgG-, IgM- and IgA levels was observed.
Negative_regulation (decrease) of Negative_regulation (decrease) of ESR in joints associated with pain
1) Confidence 0.42 Published 1978 Journal Scand. J. Rheumatol. Section Abstract Doc Link 103190 Disease Relevance 0.16 Pain Relevance 0.22
In conclusion, we hypothesize that certain types of uterine receptivity defects may be caused by the loss of appropriate ER alpha down-regulation in the mid-secretory phase, leading to defects in uterine receptivity.
Negative_regulation (regulation) of Negative_regulation (down) of ER in uterine
2) Confidence 0.38 Published 2006 Journal Reprod Biol Endocrinol Section Abstract Doc Link PMC1679803 Disease Relevance 0.29 Pain Relevance 0.24
In conclusion, we hypothesize that certain types of uterine receptivity defects may be caused by the loss of appropriate ER alpha down-regulation in the mid-secretory phase, leading to defects in uterine receptivity.
Negative_regulation (down) of Negative_regulation (regulation) of ER in uterine
3) Confidence 0.38 Published 2006 Journal Reprod Biol Endocrinol Section Abstract Doc Link PMC1679803 Disease Relevance 0.29 Pain Relevance 0.24
In conclusion, we hypothesize that certain types of uterine receptivity defects may be caused by the loss of appropriate ER alpha down-regulation in the mid-secretory phase, leading to defects in uterine receptivity.
Negative_regulation (loss) of Negative_regulation (regulation) of ER in uterine
4) Confidence 0.38 Published 2006 Journal Reprod Biol Endocrinol Section Abstract Doc Link PMC1679803 Disease Relevance 0.30 Pain Relevance 0.24
In conclusion, we hypothesize that certain types of uterine receptivity defects may be caused by the loss of appropriate ER alpha down-regulation in the mid-secretory phase, leading to defects in uterine receptivity.
Negative_regulation (loss) of Negative_regulation (down) of ER in uterine
5) Confidence 0.38 Published 2006 Journal Reprod Biol Endocrinol Section Abstract Doc Link PMC1679803 Disease Relevance 0.30 Pain Relevance 0.24
Furthermore, we hypothesize that the cancerous mammary epithelial cells sustain a defect in the ligand-dependent downregulation of ER-?
Negative_regulation (defect) of Negative_regulation (downregulation) of ER in epithelial cells
6) Confidence 0.02 Published 2004 Journal Breast Cancer Res Section Body Doc Link PMC468665 Disease Relevance 0.18 Pain Relevance 0

General Comments

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