INT82557

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Context Info
Confidence 0.41
First Reported 1999
Last Reported 2010
Negated 0
Speculated 0
Reported most in Abstract
Documents 6
Total Number 6
Disease Relevance 1.11
Pain Relevance 2.07

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

extracellular space (Csf2) extracellular region (Csf2)
Anatomy Link Frequency
spinal 2
cerebrospinal fluid 2
Csf2 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
Hyperalgesia 2 99.60 Very High Very High Very High
Pain 4 99.44 Very High Very High Very High
Nerve growth factor 12 98.32 Very High Very High Very High
algogenic 4 94.04 High High
central sensitization 4 89.52 High High
Morphine 2 88.84 High High
TRP channel 6 85.76 High High
anesthesia 5 80.80 Quite High
fibrosis 10 76.64 Quite High
Inflammation 5 75.36 Quite High
Disease Link Frequency Relevance Heat
Hyperalgesia 2 99.60 Very High Very High Very High
Pain 3 99.44 Very High Very High Very High
Stress 3 99.24 Very High Very High Very High
Fibrosis 2 76.64 Quite High
INFLAMMATION 7 75.36 Quite High
Hydrocephalus 36 71.96 Quite High
Body Weight 1 69.20 Quite High
Diabetes Mellitus 18 62.52 Quite High
Cystic Fibrosis 8 60.64 Quite High
Insulin Resistance 5 10.68 Low Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
However, it is possible that the impediment to lymphatic CSF transport may be a local phenomenon with CSF clearance increasing at other absorption sites to compensate, such that global CSF absorption is relatively unchanged.
Positive_regulation (increasing) of Localization (transport) of CSF
1) Confidence 0.41 Published 2010 Journal Cerebrospinal Fluid Res Section Body Doc Link PMC2831828 Disease Relevance 0.40 Pain Relevance 0.11
The results demonstrated that MO-induced hyperalgesia is associated with increased CSF-Glu release and that this effect is potentiated by a NGF inducer.
Positive_regulation (increased) of Localization (release) of CSF-Glu associated with hyperalgesia and nerve growth factor
2) Confidence 0.40 Published 1999 Journal Cell. Mol. Neurobiol. Section Abstract Doc Link 10384257 Disease Relevance 0.10 Pain Relevance 0.74
OBJECTIVE: Stress- and pain-related stimuli cause a release of vasopressin (AVP) and oxytocin (OT) into the cerebrospinal fluid (CSF) and extracellular fluid of the brain in various animal species.
Positive_regulation (cause) of Localization (release) of CSF in cerebrospinal fluid associated with stress and pain
3) Confidence 0.39 Published 2007 Journal Endocr Regul Section Abstract Doc Link 18257651 Disease Relevance 0.20 Pain Relevance 0.18
The results demonstrated that MO-induced hyperalgesia is associated with increased CSF-Glu release and that this effect is potentiated by a NGF inducer.
Positive_regulation (increased) of Localization (release) of CSF-Glu associated with hyperalgesia and nerve growth factor
4) Confidence 0.29 Published 1999 Journal Cell. Mol. Neurobiol. Section Abstract Doc Link 10384257 Disease Relevance 0.10 Pain Relevance 0.74
A primed-continuous ICV infusion of HN, F6AHN (total dose of 20 ug), dimerization deficient (DD) HN, STAT-3 inhibitor (75 pmol) , or artificial cerebro-spinal fluid (aCSF) was initiated at t?
Positive_regulation (initiated) of Localization (initiated) of aCSF in spinal
5) Confidence 0.09 Published 2009 Journal PLoS ONE Section Body Doc Link PMC2709436 Disease Relevance 0.25 Pain Relevance 0.07
The properties of each conductance are such that they could all play a significant role in CSF secretion.
Positive_regulation (role) of Localization (secretion) of CSF
6) Confidence 0.09 Published 2007 Journal Cerebrospinal Fluid Res Section Body Doc Link PMC2072944 Disease Relevance 0.06 Pain Relevance 0.23

General Comments

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