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Context Info
Confidence 0.20
First Reported 1999
Last Reported 1999
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0.72
Pain Relevance 0.33

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (GSR) mitochondrion (GSR) small molecule metabolic process (GSR)
oxidoreductase activity (GSR) nucleus (JUND) cytoplasm (GSR)
GSR (Homo sapiens)
JUND (Homo sapiens)
Pain Link Frequency Relevance Heat
cytokine 2 99.18 Very High Very High Very High
Inflammation 3 99.00 Very High Very High Very High
dexamethasone 1 97.24 Very High Very High Very High
cINOD 1 67.80 Quite High
Disease Link Frequency Relevance Heat
INFLAMMATION 4 99.00 Very High Very High Very High
Repression 1 88.64 High High
Asthma 4 87.16 High High
Occupational Lung Diseases 1 72.36 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Using AP-1 and NF-kappa B-dependent reporter gene assays and an immunoassay for the pro-inflammatory cytokine RANTES, we show that the over-expressed GR significantly repressed AP-1 and NF-kappa B activities in the absence of hormone and that the GC dexamethasone produced an additive inhibitory effect.
GR Negative_regulation (repressed) of AP-1 associated with inflammation, dexamethasone and cytokine
1) Confidence 0.20 Published 1999 Journal Gene Ther. Section Abstract Doc Link 10435109 Disease Relevance 0.72 Pain Relevance 0.33

General Comments

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