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Context Info
Confidence 0.22
First Reported 1999
Last Reported 1999
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0
Pain Relevance 0.78

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

Golgi apparatus (B3GAT1) carbohydrate metabolic process (B3GAT1)
Anatomy Link Frequency
cholinergic neurons 2
B3GAT1 (Homo sapiens)
Pain Link Frequency Relevance Heat
qutenza 18 99.82 Very High Very High Very High
antagonist 5 80.32 Quite High
Bile 3 75.00 Quite High
Neurotransmitter 2 74.48 Quite High
tetrodotoxin 3 46.08 Quite Low
Calcitonin gene-related peptide 1 25.00 Low Low


Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Tachykinins, probably released in small amounts by capsaicin, act by activating receptors of the NK1, NK2 and NK3 type, most probably located on intrinsic cholinergic neurons, which in turn release ACh to produce the final excitatory motor response.
Positive_regulation (activating) of NK1 Binding (receptors) of in cholinergic neurons associated with qutenza
1) Confidence 0.22 Published 1999 Journal Naunyn Schmiedebergs Arch. Pharmacol. Section Abstract Doc Link 10543438 Disease Relevance 0 Pain Relevance 0.78

General Comments

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