INT85985

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Context Info
Confidence 0.78
First Reported 1999
Last Reported 2010
Negated 1
Speculated 1
Reported most in Abstract
Documents 10
Total Number 11
Disease Relevance 6.66
Pain Relevance 3.26

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (TNNT1)
Anatomy Link Frequency
coronary artery 1
cardiac muscle cell 1
TNNT1 (Homo sapiens)
Pain Link Frequency Relevance Heat
Angina 53 99.84 Very High Very High Very High
ischemia 50 99.12 Very High Very High Very High
Percutaneous transluminal coronary angioplasty 10 98.42 Very High Very High Very High
agonist 1 81.00 Quite High
cva 9 77.60 Quite High
Pain 5 74.96 Quite High
imagery 5 60.60 Quite High
antagonist 5 50.00 Quite Low
COX-2 inhibitor 1 45.36 Quite Low
isoflurane 2 32.48 Quite Low
Disease Link Frequency Relevance Heat
Myocardial Infarction 68 99.86 Very High Very High Very High
Cv General 3 Under Development 34 99.84 Very High Very High Very High
Coronary Artery Disease 53 99.28 Very High Very High Very High
Cv Unclassified Under Development 41 99.12 Very High Very High Very High
Stable Angina Pectoris 12 98.76 Very High Very High Very High
Necrosis 21 97.44 Very High Very High Very High
Acute Coronary Syndrome 41 96.32 Very High Very High Very High
Angina 16 90.64 High High
Pathologic Constriction 1 85.24 High High
Thrombosis 2 83.28 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The purpose of this study was to determine the release of troponin T in patients with unstable and stable angina pectoris pre- and post-PTCA.
Spec (determine) Localization (release) of troponin T associated with stable angina pectoris and angina
1) Confidence 0.78 Published 1999 Journal Z Kardiol Section Abstract Doc Link 10643059 Disease Relevance 0.50 Pain Relevance 0.50
This study determines whether primary stenting produces earlier myocardial reperfusion than primary PTCA alone in patients with acute ST segment elevation myocardial infarction using troponin T release kinetics.
Localization (release) of troponin T associated with myocardial infarction and percutaneous transluminal coronary angioplasty
2) Confidence 0.78 Published 2003 Journal Angiology Section Abstract Doc Link 12678195 Disease Relevance 0.26 Pain Relevance 0.42
Relation of troponin T release kinetics to long-term clinical outcome in patients with acute ST segment elevation myocardial infarction treated with a percutaneous intervention.
Localization (release) of troponin T associated with myocardial infarction
3) Confidence 0.78 Published 2002 Journal Catheter Cardiovasc Interv Section Title Doc Link 12112882 Disease Relevance 0.33 Pain Relevance 0.12
The purpose of this study was to determine the relation of troponin T release kinetics to long-term clinical outcome in patients with an acute ST segment elevation myocardial infarction treated with a primary percutaneous intervention.
Localization (release) of troponin T associated with myocardial infarction
4) Confidence 0.78 Published 2002 Journal Catheter Cardiovasc Interv Section Abstract Doc Link 12112882 Disease Relevance 0.31 Pain Relevance 0.12
It was concluded that patients with unstable angina and no release of troponin T have less severe coronary artery disease, and have an excellent prognosis.
Neg (no) Localization (release) of troponin T in coronary artery associated with angina and coronary artery disease
5) Confidence 0.78 Published 2000 Journal Am. J. Cardiol. Section Abstract Doc Link 10758918 Disease Relevance 1.13 Pain Relevance 0.49
The question of whether to use CK-MB(mass) or troponin T or both is a subjective judgement in which the benefits of detecting and treating cases of ACS must be weighed against the costs of additional testing and management of false positives.
Localization (use) of troponin T associated with acute coronary syndrome
6) Confidence 0.73 Published 2005 Journal BMC Emerg Med Section Body Doc Link PMC1201136 Disease Relevance 1.01 Pain Relevance 0
For examples, ST changes on EKG, biologic markers such as creatine kinase (CK), CK-MB, and troponin-T, and clinical outcomes such as recovery of cardiac functions, length of hospital stay, mortality, and morbidity are frequently used.
Localization (creatine kinase) of troponin-T
7) Confidence 0.73 Published 2010 Journal Korean Journal of Anesthesiology Section Body Doc Link PMC2872846 Disease Relevance 0.64 Pain Relevance 0.24
[Release of troponin T following PTCA in patients with unstable and stable angina pectoris].
Localization (Release) of troponin T associated with stable angina pectoris and angina
8) Confidence 0.68 Published 1999 Journal Z Kardiol Section Title Doc Link 10643059 Disease Relevance 0.51 Pain Relevance 0.51
The data indicate a PTCA induced reversible ischemia of the cardiac muscle cell with additional release of the cytoplasmatic bound part of troponin T in patients with unstable angina pectoris.
Localization (release) of troponin T in cardiac muscle cell associated with angina and ischemia
9) Confidence 0.68 Published 1999 Journal Z Kardiol Section Abstract Doc Link 10643059 Disease Relevance 0.68 Pain Relevance 0.68
Mirroring unadjusted infarct size, the area under the curve (AUC) for Troponin T release was insignificantly reduced by 21% in the ADC-1004 group (ADC-1004: 2823 ± 528 vs control: 3566 ± 615, p = 0.38).
Localization (release) of Troponin T
10) Confidence 0.68 Published 2010 Journal BMC Cardiovasc Disord Section Body Doc Link PMC2955599 Disease Relevance 0.07 Pain Relevance 0.03
To assess potential confounders due to ischaemia-induced mobilisation, we subdivided the group of ACS according to the presence of troponin T.
Localization (presence) of troponin T associated with cv unclassified under development and myocardial infarction
11) Confidence 0.68 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2972200 Disease Relevance 1.21 Pain Relevance 0.15

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