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Context Info
Confidence 0.02
First Reported 2000
Last Reported 2000
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0
Pain Relevance 1.05

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

mitochondrion (Abat) plasma membrane (Flvcr2) transmembrane transport (Flvcr2)
Flvcr2 (Rattus norvegicus)
Abat (Rattus norvegicus)
Pain Link Frequency Relevance Heat
gABA 9 100.00 Very High Very High Very High
Dopamine 8 99.42 Very High Very High Very High
nMDA receptor 5 95.52 Very High Very High Very High
Action potential 2 90.64 High High
Morphine 1 84.44 Quite High
opioid receptor 1 82.36 Quite High
tetrodotoxin 1 62.16 Quite High
Glutamate 1 47.20 Quite Low


Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Moreover, our data indicate that activation of G-protein-coupled dopamine D1 receptors also induces a transporter-mediated increase in spontaneous GABA release, but through a different mechanism of action, i.e., through cAMP-dependent inhibition of Na(+)/K(+)-ATPase, inducing accumulation of intracellular sodium, reversal of the GABA carrier, and potentiation of NMDA-induced release.
transporter Positive_regulation (increase) of Localization (release) of GABA associated with gaba and dopamine
1) Confidence 0.02 Published 2000 Journal J. Neurosci. Section Abstract Doc Link 10777812 Disease Relevance 0 Pain Relevance 1.05

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