INT90348

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Context Info
Confidence 0.61
First Reported 2000
Last Reported 2010
Negated 0
Speculated 1
Reported most in Body
Documents 2
Total Number 4
Disease Relevance 1.41
Pain Relevance 0.42

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

signal transduction (Gast)
Anatomy Link Frequency
neuronal 1
stomach 1
Gast (Mus musculus)
Pain Link Frequency Relevance Heat
peptic ulcer disease 2 93.84 High High
gABA 26 87.48 High High
Somatostatin 3 83.52 Quite High
isoflurane 38 71.04 Quite High
Thalamus 4 56.00 Quite High
Nicotine 2 54.68 Quite High
Limbic system 2 51.40 Quite High
Analgesic 4 46.72 Quite Low
halothane 38 38.40 Quite Low
Hippocampus 2 35.92 Quite Low
Disease Link Frequency Relevance Heat
Frailty 2 100.00 Very High Very High Very High
Helicobacter Infection 3 98.48 Very High Very High Very High
Infection 5 95.76 Very High Very High Very High
Targeted Disruption 19 95.04 Very High Very High Very High
Ulcers 4 93.84 High High
Disease 2 93.32 High High
Stomach Cancer 2 92.88 High High
Depression 2 15.80 Low Low
Unconsciousness 8 5.00 Very Low Very Low Very Low
Sleep Disorders 4 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Altered gastrin regulation in mice infected with Helicobacter felis.
Regulation (regulation) of gastrin
1) Confidence 0.61 Published 2000 Journal Dig. Dis. Sci. Section Title Doc Link 10961708 Disease Relevance 0.77 Pain Relevance 0.20
In the present study, we examined the long-term effect of G-gly on the atrophy-promoting effects of amidated gastrin in the mouse stomach with or without Helicobacter infection.
Spec (examined) Regulation (effect) of gastrin in stomach associated with helicobacter infection and frailty
2) Confidence 0.45 Published 2004 Journal Cancer Res. Section Abstract Doc Link 15548680 Disease Relevance 0.63 Pain Relevance 0
The family of GABAA receptors is responsible for the majority of fast neuronal inhibition in the mammalian CNS, and is thought to be a target of GAs.
Regulation (target) of GAs in neuronal
3) Confidence 0.08 Published 2010 Journal British Journal of Pharmacology Section Body Doc Link PMC2989583 Disease Relevance 0 Pain Relevance 0.08
These results are summarized in Table 1, and they show that the GAs acting on the GABAA receptor could be divided into several groups, depending on their binding site.
Regulation (acting) of GAs
4) Confidence 0.08 Published 2010 Journal British Journal of Pharmacology Section Body Doc Link PMC2989583 Disease Relevance 0 Pain Relevance 0.13

General Comments

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