INT92423

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Context Info
Confidence 0.02
First Reported 2000
Last Reported 2000
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 2
Disease Relevance 0.28
Pain Relevance 0.86

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

peptidase activity (Mme) plasma membrane (Mme)
Anatomy Link Frequency
brain 2
EC3 (Mus musculus)
Mme (Mus musculus)
Pain Link Frequency Relevance Heat
Enkephalin 12 100.00 Very High Very High Very High
Opioid 4 83.76 Quite High
analgesia 2 82.40 Quite High
Endogenous opioid 2 75.00 Quite High
Eae 2 25.00 Low Low
Kappa opioid receptor 2 25.00 Low Low
Disease Link Frequency Relevance Heat
Targeted Disruption 6 80.72 Quite High
Aggression 2 25.00 Low Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The biological inactivation of enkephalins by neutral endopeptidase (enkephalinase, NEP, EC3.4.24.11) represents a major mechanism for the termination of enkephalinergic signalling in brain.
EC3 Negative_regulation (inactivation) of enkephalinase in brain associated with enkephalin
1) Confidence 0.02 Published 2000 Journal Regul. Pept. Section Abstract Doc Link 11102652 Disease Relevance 0.14 Pain Relevance 0.43
The biological inactivation of enkephalins by neutral endopeptidase (enkephalinase, NEP, EC3.4.24.11) represents a major mechanism for the termination of enkephalinergic signalling in brain.
EC3 Negative_regulation (inactivation) of NEP in brain associated with enkephalin
2) Confidence 0.02 Published 2000 Journal Regul. Pept. Section Abstract Doc Link 11102652 Disease Relevance 0.14 Pain Relevance 0.43

General Comments

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