INT92876

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Context Info
Confidence 0.45
First Reported 2000
Last Reported 2000
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 2
Disease Relevance 0.71
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (PDE5A) signal transduction (PDE5A) cellular_component (PDE5A)
PDE5A (Homo sapiens)
Pain Link Frequency Relevance Heat
Angina 2 39.28 Quite Low
ischemia 2 25.00 Low Low
Disease Link Frequency Relevance Heat
Coronary Heart Disease 4 97.92 Very High Very High Very High
Cardiovascular Disease 2 86.92 High High
Reprotox - General 2 6 85.08 High High
Myocardial Infarction 6 41.52 Quite Low
Cv General 3 Under Development 2 39.28 Quite Low
Stroke 2 37.92 Quite Low
Heart Disease 2 25.00 Low Low
Infarction 2 25.00 Low Low
Cv Unclassified Under Development 2 25.00 Low Low
Stress 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Sildenafil is a potent and selective inhibitor of phosphodiesterase type 5 (PDE5), the enzyme responsible for the degradation of cyclic guanosine monophosphate (cGMP).
Regulation (responsible) of Protein_catabolism (degradation) of PDE5
1) Confidence 0.45 Published 2000 Journal Am. J. Med. Section Abstract Doc Link 11137498 Disease Relevance 0.35 Pain Relevance 0
Sildenafil is a potent and selective inhibitor of phosphodiesterase type 5 (PDE5), the enzyme responsible for the degradation of cyclic guanosine monophosphate (cGMP).
Regulation (responsible) of Protein_catabolism (degradation) of phosphodiesterase type 5
2) Confidence 0.39 Published 2000 Journal Am. J. Med. Section Abstract Doc Link 11137498 Disease Relevance 0.35 Pain Relevance 0

General Comments

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