INT93787

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Context Info
Confidence 0.26
First Reported 2000
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 6
Total Number 10
Disease Relevance 5.81
Pain Relevance 0.67

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

Anatomy Link Frequency
muscles 5
blood 1
Tnnc1 (Mus musculus)
Pain Link Frequency Relevance Heat
ischemia 51 99.16 Very High Very High Very High
Percutaneous transluminal coronary angioplasty 10 97.32 Very High Very High Very High
fibrosis 10 86.40 High High
Angina 4 72.40 Quite High
cytokine 50 55.72 Quite High
Inflammation 25 55.32 Quite High
isoflurane 3 50.00 Quite Low
Opioid 2 50.00 Quite Low
anesthesia 10 5.00 Very Low Very Low Very Low
Inflammatory response 5 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Reperfusion Injury 230 99.40 Very High Very High Very High
Fatigue 40 99.30 Very High Very High Very High
Injury 289 99.28 Very High Very High Very High
Cv Unclassified Under Development 85 99.16 Very High Very High Very High
Death 45 94.72 High High
Parkinson's Disease 5 89.84 High High
Fibrosis 10 86.40 High High
Cv General 3 Under Development 2 72.40 Quite High
Necrosis 10 57.36 Quite High
INFLAMMATION 30 55.32 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
CONCLUSIONS: In this study, the use of isoflurane-sufentanil in comparison with propofol-sufentanil anaesthesia does not afford additional reduction of postoperative cTnI levels.


Negative_regulation (reduction) of cTnI
1) Confidence 0.26 Published 2010 Journal Br J Anaesth Section Body Doc Link 20573633 Disease Relevance 0 Pain Relevance 0
Two other AMI cases document the significant cTnI decline in results after peak values.
Negative_regulation (decline) of cTnI
2) Confidence 0.23 Published 2009 Journal Clin. Chim. Acta Section Body Doc Link 19135041 Disease Relevance 0.07 Pain Relevance 0
The purpose of the study was to measure cardiac troponin I (cTnI) and cardiac troponin T (cTnT) levels after elective uncomplicated successful percutaneous transluminal coronary angioplasty (PTCA) with or without coronary stenting and to compare their results with serum creatinine kinase MB isoenzyme (CKMB).
Negative_regulation (measure) of cTnI associated with percutaneous transluminal coronary angioplasty
3) Confidence 0.22 Published 2001 Journal Catheter Cardiovasc Interv Section Abstract Doc Link 11387602 Disease Relevance 0.34 Pain Relevance 0.20
CONCLUSIONS: The cTnI release post ExT is more frequently observed in patients with stable CAD with ejection fraction < or = 50%, low physical activity, and max systolic blood pressure > 160 mm Hg at peak ExT.
Negative_regulation (post) of cTnI in blood
4) Confidence 0.19 Published 2010 Journal Kardiol Pol Section Body Doc Link 20425701 Disease Relevance 0 Pain Relevance 0
The purpose of the study was to measure cardiac troponin I (cTnI) and cardiac troponin T (cTnT) levels after apparently successful percutaneous transluminal coronary angioplasty (PTCA) with or without coronary stenting and to compare their results with serum creatine kinase (CK) and its isoform, creatine kinase-MB (CKMB).
Negative_regulation (measure) of cTnI associated with percutaneous transluminal coronary angioplasty
5) Confidence 0.09 Published 2000 Journal Med. Sci. Monit. Section Abstract Doc Link 11208396 Disease Relevance 0.34 Pain Relevance 0.24
By 14 days, absolute and specific maximal forces and fatigue resistance in ischemic/reperfused soleus muscles were still reduced (?
Negative_regulation (reduced) of muscles in muscles associated with fatigue
6) Confidence 0.05 Published 2010 Journal Journal of Biomedicine and Biotechnology Section Abstract Doc Link PMC2866363 Disease Relevance 0.53 Pain Relevance 0.08
In contrast to Carvalho et al. [7], Fish et al. [8] found that full muscle recovery was not reached 42 days after 2 hours of ischemia since maximal force production was still reduced in ischemic-reperfused muscles as compared to control muscles in rats.
Negative_regulation (reduced) of muscles in muscles associated with ischemia
7) Confidence 0.04 Published 2010 Journal Journal of Biomedicine and Biotechnology Section Body Doc Link PMC2866363 Disease Relevance 1.77 Pain Relevance 0.11
By 14 days, absolute maximal force (P0, Figure 2), specific maximal force (P0/m, Figure 3), fatigue resistance (FR, Figure 4) and muscle weight (Figure 5) were still markedly reduced in ischemic/reperfused muscles as compared to control muscles (P < .05).
Negative_regulation (reduced) of muscles in muscles associated with fatigue
8) Confidence 0.04 Published 2010 Journal Journal of Biomedicine and Biotechnology Section Body Doc Link PMC2866363 Disease Relevance 1.04 Pain Relevance 0.04
However, specific maximal force (Figure 3) was still reduced in ischemic/reperfused muscles as compared to control muscles, by day 56 (P < .05).
Negative_regulation (reduced) of muscles in muscles
9) Confidence 0.04 Published 2010 Journal Journal of Biomedicine and Biotechnology Section Body Doc Link PMC2866363 Disease Relevance 0.48 Pain Relevance 0
Our results indicate that maximal force and fatigue resistance in ischemic-reperfused muscles were still markedly reduced 14 days after IR injury, in agreement with previous observations [5, 21].
Negative_regulation (reduced) of muscles in muscles associated with reperfusion injury, injury and fatigue
10) Confidence 0.04 Published 2010 Journal Journal of Biomedicine and Biotechnology Section Body Doc Link PMC2866363 Disease Relevance 1.25 Pain Relevance 0

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