INT94088

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Context Info
Confidence 0.55
First Reported 2001
Last Reported 2008
Negated 0
Speculated 0
Reported most in Abstract
Documents 2
Total Number 2
Disease Relevance 0.39
Pain Relevance 0.53

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

signal transduction (Pde6b)
Anatomy Link Frequency
PMN 1
Pde6b (Mus musculus)
Pain Link Frequency Relevance Heat
orphanin 8 96.76 Very High Very High Very High
Pain 1 94.80 High High
opioid receptor 1 94.08 High High
aspirin 4 75.00 Quite High
Inflammation 1 38.72 Quite Low
Central nervous system 3 5.00 Very Low Very Low Very Low
anesthesia 2 5.00 Very Low Very Low Very Low
Buprenorphine 1 5.00 Very Low Very Low Very Low
imagery 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Cytomegalovirus Infection 9 95.60 Very High Very High Very High
Pain 1 94.80 High High
Infection 20 5.00 Very Low Very Low Very Low
Viral Infection 7 5.00 Very Low Very Low Very Low
Sprains And Strains 5 5.00 Very Low Very Low Very Low
Death 3 5.00 Very Low Very Low Very Low
Disease 3 5.00 Very Low Very Low Very Low
Spontaneous Abortion 2 5.00 Very Low Very Low Very Low
Ganglion Cysts 2 5.00 Very Low Very Low Very Low
Cataract 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The following targeting sequence was used for the PDE6b shRNA construct: 5?
Transcription (used) of PDE6b
1) Confidence 0.55 Published 2008 Journal Developmental Dynamics Section Body Doc Link PMC2532702 Disease Relevance 0.29 Pain Relevance 0
Human PMN express mRNA for Noci-R, its nucleotide sequence determined, and specific binding with [(125)I]-labeled nociceptin gave an apparent K(d) approximately 1.5 nM for this PMN opioid receptor.
Transcription (express) of Noci-R in PMN associated with orphanin and opioid receptor
2) Confidence 0.04 Published 2001 Journal J. Immunol. Section Abstract Doc Link 11238602 Disease Relevance 0.09 Pain Relevance 0.53

General Comments

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