INT94535

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Context Info
Confidence 0.34
First Reported 2001
Last Reported 2010
Negated 0
Speculated 1
Reported most in Body
Documents 1
Total Number 2
Disease Relevance 1.70
Pain Relevance 0.07

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (MT2A) nucleus (MT2A) cytoplasm (MT2A)
Anatomy Link Frequency
MT2 2
bladder 2
MT2A (Homo sapiens)
Pain Link Frequency Relevance Heat
interstitial cystitis 1 69.44 Quite High
Bile 2 5.00 Very Low Very Low Very Low
Inflammation 1 5.00 Very Low Very Low Very Low
abdominal pain 1 5.00 Very Low Very Low Very Low
headache 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Bladder Cancer 9 99.80 Very High Very High Very High
Opisthorchiasis 3 92.40 High High
Infection 64 90.44 High High
Trematode Infection 2 87.40 High High
Clonorchiasis 38 83.76 Quite High
Bladder Disease 1 72.64 Quite High
Cystitis 1 70.48 Quite High
Interstitial Cystitis 1 69.44 Quite High
Carcinoma In Situ 1 60.40 Quite High
Cancer 2 43.36 Quite Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The goals of this study were to determine the expression of metallothionein isoform 1 and 2 proteins (MT-1 and MT-2) in bladder cancer and then to determine which MT isoform-specific genes promoted the expression of these proteins.
Spec (determine) Negative_regulation (determine) of Spec (determine) Gene_expression (expression) of MT-2 in bladder associated with bladder cancer
1) Confidence 0.34 Published 2001 Journal Cancer Detect. Prev. Section Abstract Doc Link 11270423 Disease Relevance 0.56 Pain Relevance 0.07
The relative risks were significantly lower in the MT2 and ST6M groups in the heavy endemic areas and in all groups in moderate endemic areas.
Negative_regulation (lower) of Gene_expression (groups) of MT2 in MT2
2) Confidence 0.13 Published 2010 Journal PLoS Neglected Tropical Diseases Section Body Doc Link PMC2821909 Disease Relevance 1.14 Pain Relevance 0

General Comments

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