INT9484

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Context Info
Confidence 0.78
First Reported 1990
Last Reported 2011
Negated 2
Speculated 14
Reported most in Abstract
Documents 175
Total Number 189
Disease Relevance 34.89
Pain Relevance 68.54

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (Jun) aging (Jun) nucleus (Jun)
DNA binding (Jun) transcription factor binding (Jun)
Anatomy Link Frequency
neurons 14
spinal cord 11
nucleus 9
brain 8
vagus nerve 6
Jun (Rattus norvegicus)
Pain Link Frequency Relevance Heat
Pain 116 100.00 Very High Very High Very High
Hippocampus 86 100.00 Very High Very High Very High
Nerve growth factor 75 100.00 Very High Very High Very High
Thalamus 64 100.00 Very High Very High Very High
c fibre 54 100.00 Very High Very High Very High
Opioid 51 100.00 Very High Very High Very High
nociceptor 30 100.00 Very High Very High Very High
opiate 13 100.00 Very High Very High Very High
metalloproteinase 9 100.00 Very High Very High Very High
Neuronal nitric oxide synthase 9 100.00 Very High Very High Very High
Disease Link Frequency Relevance Heat
Shock 7 100.00 Very High Very High Very High
Lumbar Puncture Related Headaches 2 99.84 Very High Very High Very High
Ganglion Cysts 89 99.74 Very High Very High Very High
Stress 63 99.62 Very High Very High Very High
Hypotension 83 99.44 Very High Very High Very High
Death 58 99.38 Very High Very High Very High
Injury 184 99.30 Very High Very High Very High
Benign Prostatic Hypertrophy 8 99.28 Very High Very High Very High
Coronary Heart Disease 24 99.22 Very High Very High Very High
Cv Unclassified Under Development 136 99.16 Very High Very High Very High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Therefore, expression and DNA binding activity of the AP-1 transcription factors are correlated with opioid peptide gene expression in the rat hippocampus.
Gene_expression (expression) of AP-1 in hippocampus associated with hippocampus and opioid
1) Confidence 0.78 Published 1993 Journal J. Neurochem. Section Abstract Doc Link 8417141 Disease Relevance 0.09 Pain Relevance 0.61
Kainate-induced changes in opioid peptide genes and AP-1 protein expression in the rat hippocampus.
Gene_expression (expression) of AP-1 in hippocampus associated with hippocampus and opioid
2) Confidence 0.78 Published 1993 Journal J. Neurochem. Section Title Doc Link 8417141 Disease Relevance 0.07 Pain Relevance 0.59
Transection of the cervical sympathetic trunk induced expression of c-JUN and JUN D, but not of JUN B, in the preganglionic sympathetic neurons of thoracic spinal cord.
Gene_expression (expression) of JUN in sympathetic associated with spinal cord
3) Confidence 0.77 Published 1991 Journal Neuroscience Section Abstract Doc Link 1762686 Disease Relevance 0.13 Pain Relevance 0.37
Vagotomy induced the expression of c-JUN and JUN D in the nodose ganglion, dorsal motor nucleus of the vagus nerve and nucleus ambiguus, whereas JUN B was not expressed in these areas, c-JUN and JUN D appeared after 10 h in the nodose ganglion and after 24 h in the dorsal motor nucleus of the vagus nerve with a maximum of immunoreactivity after 48 h.
Gene_expression (expression) of JUN in nucleus associated with ganglion cysts and vagus nerve
4) Confidence 0.77 Published 1991 Journal Neuroscience Section Abstract Doc Link 1762686 Disease Relevance 0.19 Pain Relevance 0.21
Similar to vagotomy, application of colchicine and vinblastine on to the intact vagus nerve induced expression of c-JUN and JUN D.
Gene_expression (expression) of JUN in vagus nerve associated with vagus nerve
5) Confidence 0.77 Published 1991 Journal Neuroscience Section Abstract Doc Link 1762686 Disease Relevance 0.15 Pain Relevance 0.38
We postulate that the expression of c-JUN and JUN D in axotomized neurons is induced by deprivation of a target-derived suppressor.
Gene_expression (expression) of JUN in neurons
6) Confidence 0.77 Published 1991 Journal Neuroscience Section Abstract Doc Link 1762686 Disease Relevance 0 Pain Relevance 0.24
In these neurons, expression of c-JUN was still enhanced after 60 days whereas JUN D had returned to basal level.
Gene_expression (expression) of JUN in neurons
7) Confidence 0.77 Published 1991 Journal Neuroscience Section Abstract Doc Link 1762686 Disease Relevance 0.12 Pain Relevance 0.35
Crush of the vagus nerve initially evoked an intense expression of c-JUN and JUN D, but in the course of regeneration the expression of c-JUN and JUN D had returned to more basal levels after 100 days.
Gene_expression (expression) of JUN in vagus nerve associated with vagus nerve
8) Confidence 0.77 Published 1991 Journal Neuroscience Section Abstract Doc Link 1762686 Disease Relevance 0.17 Pain Relevance 0.36
Similar to vagotomy, application of colchicine and vinblastine on to the intact vagus nerve induced expression of c-JUN and JUN D.
Gene_expression (expression) of JUN in vagus nerve associated with vagus nerve
9) Confidence 0.77 Published 1991 Journal Neuroscience Section Abstract Doc Link 1762686 Disease Relevance 0.15 Pain Relevance 0.38
We postulate that the expression of c-JUN and JUN D in axotomized neurons is induced by deprivation of a target-derived suppressor.
Gene_expression (expression) of JUN in neurons
10) Confidence 0.77 Published 1991 Journal Neuroscience Section Abstract Doc Link 1762686 Disease Relevance 0 Pain Relevance 0.24
Expression of c-JUN, JUN B and JUN D proteins was investigated in axotomized neurons following transection of the vagus nerve and the cervical sympathetic trunk in the rat.
Spec (investigated) Gene_expression (Expression) of JUN in neurons associated with vagus nerve
11) Confidence 0.77 Published 1991 Journal Neuroscience Section Abstract Doc Link 1762686 Disease Relevance 0.15 Pain Relevance 0.16
Crush of the vagus nerve initially evoked an intense expression of c-JUN and JUN D, but in the course of regeneration the expression of c-JUN and JUN D had returned to more basal levels after 100 days.
Gene_expression (expression) of JUN in vagus nerve associated with vagus nerve
12) Confidence 0.77 Published 1991 Journal Neuroscience Section Abstract Doc Link 1762686 Disease Relevance 0.16 Pain Relevance 0.36
One hour after vagotomy, c-JUN and JUN B were transynaptically expressed in the area of central termination of sensory vagal neurons and declined within 10 h to basal levels.
Gene_expression (expressed) of JUN in neurons
13) Confidence 0.77 Published 1991 Journal Neuroscience Section Abstract Doc Link 1762686 Disease Relevance 0.11 Pain Relevance 0.30
Vagotomy induced the expression of c-JUN and JUN D in the nodose ganglion, dorsal motor nucleus of the vagus nerve and nucleus ambiguus, whereas JUN B was not expressed in these areas, c-JUN and JUN D appeared after 10 h in the nodose ganglion and after 24 h in the dorsal motor nucleus of the vagus nerve with a maximum of immunoreactivity after 48 h.
Gene_expression (expression) of JUN in nucleus associated with ganglion cysts and vagus nerve
14) Confidence 0.77 Published 1991 Journal Neuroscience Section Abstract Doc Link 1762686 Disease Relevance 0.19 Pain Relevance 0.21
Expression of c-JUN, JUN B and JUN D proteins in rat nervous system following transection of vagus nerve and cervical sympathetic trunk.
Gene_expression (Expression) of JUN in nervous system associated with vagus nerve
15) Confidence 0.77 Published 1991 Journal Neuroscience Section Title Doc Link 1762686 Disease Relevance 0.17 Pain Relevance 0.29
Transection of the cervical sympathetic trunk induced expression of c-JUN and JUN D, but not of JUN B, in the preganglionic sympathetic neurons of thoracic spinal cord.
Gene_expression (expression) of JUN in sympathetic associated with spinal cord
16) Confidence 0.77 Published 1991 Journal Neuroscience Section Abstract Doc Link 1762686 Disease Relevance 0.13 Pain Relevance 0.37
The c-JUN protein remained expressed at an increased level up to 100 days, whereas the immunoreactivity of JUN D declined after five days.
Gene_expression (expressed) of JUN
17) Confidence 0.77 Published 1991 Journal Neuroscience Section Abstract Doc Link 1762686 Disease Relevance 0.18 Pain Relevance 0.32
Expression of c-JUN, JUN B and JUN D proteins was investigated in axotomized neurons following transection of the vagus nerve and the cervical sympathetic trunk in the rat.
Spec (investigated) Gene_expression (Expression) of JUN in neurons associated with vagus nerve
18) Confidence 0.77 Published 1991 Journal Neuroscience Section Abstract Doc Link 1762686 Disease Relevance 0.15 Pain Relevance 0.16
Vagotomy induced the expression of c-JUN and JUN D in the nodose ganglion, dorsal motor nucleus of the vagus nerve and nucleus ambiguus, whereas JUN B was not expressed in these areas, c-JUN and JUN D appeared after 10 h in the nodose ganglion and after 24 h in the dorsal motor nucleus of the vagus nerve with a maximum of immunoreactivity after 48 h.
Neg (not) Gene_expression (expressed) of JUN in nucleus associated with ganglion cysts and vagus nerve
19) Confidence 0.77 Published 1991 Journal Neuroscience Section Abstract Doc Link 1762686 Disease Relevance 0.20 Pain Relevance 0.24
Expression of c-JUN, JUN B and JUN D proteins in rat nervous system following transection of vagus nerve and cervical sympathetic trunk.
Gene_expression (Expression) of JUN in nervous system associated with vagus nerve
20) Confidence 0.77 Published 1991 Journal Neuroscience Section Title Doc Link 1762686 Disease Relevance 0.17 Pain Relevance 0.29

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