INT9545

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Context Info
Confidence 0.04
First Reported 1991
Last Reported 1992
Negated 0
Speculated 1
Reported most in Abstract
Documents 2
Total Number 2
Disease Relevance 1.84
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

extracellular region (F13A1) transferase activity, transferring acyl groups (F13A1) cytoplasm (F13A1)
F13A1 (Homo sapiens)
F2 (Sus scrofa)
Pain Term Frequency Confidence Heat
Angina 2 40.28 Quite Low
Disease Term Frequency Confidence Heat
Thrombosis 15 99.32 Very High Very High Very High
Rupture 7 88.08 High High
Coronary Artery Disease 1 76.04 Quite High
Atherosclerotic Plaque 3 75.00 Quite High
Increased Venous Pressure Under Development 1 73.08 Quite High
Syndrome 2 61.84 Quite High
Pathologic Constriction 2 43.92 Quite Low
Coronary Heart Disease 2 42.60 Quite Low
Myocardial Infarction 2 41.28 Quite Low
Cv General 3 Under Development 2 40.28 Quite Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
After spontaneous or pharmacological reperfusion, the surface of the residual thrombus is very thrombogenic and may contribute to reocclusion; this is partially due to thrombin bound to fibrin in the original thrombus. 4) Systemic factors.
fibrin Spec (partially) Binding (bound) of thrombin associated with thrombosis
1) Confidence 0.04 Published 1991 Journal Drugs Section Abstract Doc Link 1726211 Disease Relevance 0.92 Pain Relevance 0
Using recombinant hirudin as a pharmacological tool in our flow studies, we observed that rethrombosis is partially caused by thrombin bound to fibrin in the original thrombus, because the effect is abolished by the specific thrombin inhibitor.
fibrin Binding (bound) of thrombin associated with thrombosis
2) Confidence 0.02 Published 1992 Journal Circulation Section Abstract Doc Link 1424053 Disease Relevance 0.92 Pain Relevance 0

General Comments

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