INT95638

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Context Info
Confidence 0.44
First Reported 2001
Last Reported 2001
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 2
Disease Relevance 3.08
Pain Relevance 0.20

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

aging (FAS) protein complex assembly (FAS) cytoplasm (FAS)
signal transducer activity (FAS) cytosol (FAS) signal transduction (FAS)
Anatomy Link Frequency
B-cell 4
FAS (Homo sapiens)
Pain Link Frequency Relevance Heat
cytokine 2 99.32 Very High Very High Very High
Inflammatory response 2 98.76 Very High Very High Very High
alcohol 2 41.12 Quite Low
Paracetamol 2 38.08 Quite Low
ischemia 2 25.36 Quite Low
Disease Link Frequency Relevance Heat
Apoptosis 26 99.46 Very High Very High Very High
Necrosis 4 99.24 Very High Very High Very High
Cancer 2 98.60 Very High Very High Very High
INFLAMMATION 2 98.58 Very High Very High Very High
Lymphatic System Cancer 2 91.44 High High
Disease 8 75.00 Quite High
Liver Disease 4 66.00 Quite High
Autoimmune Hepatitis 2 57.92 Quite High
Metabolic Disorder 2 54.08 Quite High
Hepatocellular Cancer 2 46.64 Quite Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Apoptosis, or programmed cell death, and the elimination of apoptotic cells are crucial factors in the maintenance of liver health Apoptosis allows hepatocytes to die without provoking a potentially harmful inflammatory response In contrast to necrosis, apoptosis is tightly controlled and regulated via several mechanisms, including Fas/Fas ligand interactions, the effects of cytokines such as tumor necrosis factor alpha (TNF-alpha) and transforming growth factor beta (TGF-beta), and the influence of pro- and antiapoptotic mitochondria-associated proteins of the B-cell lymphoma-2 (Bcl-2) family.
Regulation (regulated) of Fas Binding (interactions) of in B-cell associated with necrosis, inflammatory response, lymphatic system cancer, cancer, apoptosis and cytokine
1) Confidence 0.44 Published 2001 Journal Crit Rev Clin Lab Sci Section Abstract Doc Link 11347718 Disease Relevance 1.54 Pain Relevance 0.10
Apoptosis, or programmed cell death, and the elimination of apoptotic cells are crucial factors in the maintenance of liver health Apoptosis allows hepatocytes to die without provoking a potentially harmful inflammatory response In contrast to necrosis, apoptosis is tightly controlled and regulated via several mechanisms, including Fas/Fas ligand interactions, the effects of cytokines such as tumor necrosis factor alpha (TNF-alpha) and transforming growth factor beta (TGF-beta), and the influence of pro- and antiapoptotic mitochondria-associated proteins of the B-cell lymphoma-2 (Bcl-2) family.
Regulation (regulated) of Fas Binding (interactions) of in B-cell associated with necrosis, inflammatory response, lymphatic system cancer, cancer, apoptosis and cytokine
2) Confidence 0.44 Published 2001 Journal Crit Rev Clin Lab Sci Section Abstract Doc Link 11347718 Disease Relevance 1.54 Pain Relevance 0.10

General Comments

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