INT97774

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Context Info
Confidence 0.32
First Reported 2001
Last Reported 2008
Negated 0
Speculated 0
Reported most in Abstract
Documents 2
Total Number 2
Disease Relevance 0.83
Pain Relevance 1.45

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

plasma membrane (Gpr18) signal transducer activity (Gpr18)
Anatomy Link Frequency
brain 1
Gpr18 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
IPN 1 99.12 Very High Very High Very High
antagonist 1 98.56 Very High Very High Very High
allodynia 3 97.32 Very High Very High Very High
Cannabinoid 1 97.08 Very High Very High Very High
Sciatic nerve 1 92.80 High High
agonist 2 88.84 High High
Cannabinoid receptor 2 88.52 High High
intrathecal 1 85.80 High High
Neuropathic pain 4 85.28 High High
Analgesic 2 84.40 Quite High
Disease Link Frequency Relevance Heat
Inflammatory Pain 1 99.12 Very High Very High Very High
Neuropathic Pain 7 97.32 Very High Very High Very High
Pain 4 78.56 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The biosynthesis of NAGly and its degradation by the enzyme fatty acid amide hydrolase can be observed in rat brain tissue.
Protein_catabolism (degradation) of NAGly in brain
1) Confidence 0.32 Published 2001 Journal J. Biol. Chem. Section Abstract Doc Link 11518719 Disease Relevance 0.25 Pain Relevance 0.32
The NAGly degradation products, arachidonic acid and glycine (700 nmol), did not reduce allodynia.
Protein_catabolism (degradation) of NAGly associated with allodynia
2) Confidence 0.23 Published 2008 Journal Neuropharmacology Section Abstract Doc Link 17588618 Disease Relevance 0.58 Pain Relevance 1.13

General Comments

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