INT105257

Context Info Confidence 0.23 First Reported 2002 Last Reported 2002 Negated 0 Speculated 0 Reported most in Abstract Documents 1 Total Number 1 Disease Relevance 0 Pain Relevance 0.26

plasma membrane (F2rl1) signal transducer activity (F2rl1)

Anatomy Link Frequency sensory neurons 2 F2rl1 (Rattus norvegicus)

Pain Link Frequency Relevance Heat qutenza 2 96.80 agonist 2 83.80

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Sentences Mentioned In

Key:

Protein

Mutation

Event

Anatomy

Negation

Speculation

Pain term

Disease term

In conclusion, our study demonstrates expression of PAR-2 in rat pancreatic acini as well as parotid acini and indicates that nitric oxide participates in the PAR-2-mediated in vivo secretion of pancreatic amylase, and, to a certain extent, of salivary amylase, although capsaicin-sensitive sensory neurons, known to be activated by PAR-2, are not involved in the evoked pancreatic or salivary amylase secretion.

Regulation (participates) of Localization (secretion) of PAR-2-mediated in sensory neurons associated with qutenza

1) Confidence 0.23 Published 2002 Journal Life Sci. Section Abstract Doc Link 12231404 Disease Relevance 0 Pain Relevance 0.26

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