INT127152

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Context Info
Confidence 0.22
First Reported 2005
Last Reported 2005
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 2
Disease Relevance 0.52
Pain Relevance 0.07

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

mitochondrion (Acaa2) lipid metabolic process (Acaa2)
Acaa2 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
cerebral cortex 4 75.00 Quite High
anesthesia 2 25.00 Low Low
Disease Link Frequency Relevance Heat
Ketoacidosis 2 98.64 Very High Very High Very High
Intellectual Impairment 2 62.56 Quite High
Convulsion 2 60.72 Quite High
Acid Base Imbalance 2 52.24 Quite High
Vomiting 2 50.64 Quite High
Coma 2 49.60 Quite Low
Lactic Acidosis 2 38.24 Quite Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Biochemically, beta-ketothiolase deficiency is characterized by intermittent ketoacidosis and urinary excretion of 2-methyl-acetoacetate (MAA), 2-methyl-3-hydroxybutyrate (MHB) and tiglylglycine (TG), whereas in MHBD deficiency only MHB and tiglylglycine accumulate.
Negative_regulation (deficiency) of beta-ketothiolase associated with ketoacidosis
1) Confidence 0.22 Published 2005 Journal J. Inherit. Metab. Dis. Section Abstract Doc Link 15902553 Disease Relevance 0.32 Pain Relevance 0.04
Mitochondrial beta-ketothiolase and 2-methyl-3-hydroxybutyryl-CoA dehydrogenase (MHBD) deficiencies are inherited neurometabolic disorders affecting isoleucine catabolism.
Negative_regulation (deficiencies) of beta-ketothiolase
2) Confidence 0.22 Published 2005 Journal J. Inherit. Metab. Dis. Section Abstract Doc Link 15902553 Disease Relevance 0.19 Pain Relevance 0.04

General Comments

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