INT147407

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Context Info
Confidence 0.91
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 2
Disease Relevance 0.33
Pain Relevance 1.06

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

signal transduction (FGF2) extracellular space (FGF2) extracellular region (FGF2)
nucleus (FGF2) cytoplasm (FGF2)
Anatomy Link Frequency
neural 2
FGF2 (Homo sapiens)
Pain Link Frequency Relevance Heat
substance P 16 98.48 Very High Very High Very High
metalloproteinase 2 86.72 High High
Inflammation 2 86.24 High High
Neuropeptide 2 80.92 Quite High
Pain 2 80.04 Quite High
Disease Link Frequency Relevance Heat
INFLAMMATION 2 86.24 High High
Pain 2 80.04 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
We further examined if catabolic growth factors such as basic fibroblast growth factor (bFGF or FGF-2) or IL-1beta accelerate matrix degradation via a neural pathway upregulation of substance P and NK(1)-R.
Protein_catabolism (degradation) of FGF-2 in neural associated with substance p
1) Confidence 0.91 Published 2008 Journal J. Cell. Physiol. Section Abstract Doc Link 17960584 Disease Relevance 0.17 Pain Relevance 0.53
We further examined if catabolic growth factors such as basic fibroblast growth factor (bFGF or FGF-2) or IL-1beta accelerate matrix degradation via a neural pathway upregulation of substance P and NK(1)-R.
Protein_catabolism (degradation) of bFGF in neural associated with substance p
2) Confidence 0.81 Published 2008 Journal J. Cell. Physiol. Section Abstract Doc Link 17960584 Disease Relevance 0.17 Pain Relevance 0.53

General Comments

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