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Context Info
Confidence 0.12
First Reported 2009
Last Reported 2009
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0.27
Pain Relevance 0.86

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

nucleoplasm (JUN) nuclear chromosome (JUN) aging (JUN)
small molecule metabolic process (POMC) DNA binding (JUN) cell-cell signaling (POMC)
Anatomy Link Frequency
T lymphocytes 2
POMC (Homo sapiens)
JUN (Homo sapiens)
Pain Link Frequency Relevance Heat
Endogenous opioid 1 99.98 Very High Very High Very High
Morphine 3 99.46 Very High Very High Very High
Opioid 6 94.08 High High
Pain 1 83.12 Quite High
mu opioid receptor 1 38.24 Quite Low
Disease Link Frequency Relevance Heat
Acquired Immune Deficiency Syndrome Or Hiv Infection 1 92.40 High High
Cancer 1 92.08 High High
Pain 1 83.12 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
We demonstrated that morphine and the endogenous opioid beta-endorphin inhibited the transcription of IL-2 in activated human T lymphocytes as well as the activation of the transcription factors AP-1, NFAT, and NF-kappaB, which transactivate IL-2.
beta-endorphin Negative_regulation (inhibited) of Positive_regulation (activation) of AP-1 in T lymphocytes associated with endogenous opioid and morphine
1) Confidence 0.12 Published 2009 Journal J. Immunol. Section Abstract Doc Link 19561113 Disease Relevance 0.27 Pain Relevance 0.86

General Comments

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