INT168394

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Context Info
Confidence 0.37
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0.82
Pain Relevance 0.74

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

signal transduction (Cnr2) plasma membrane (Cnr2) signal transducer activity (Cnr2)
Anatomy Link Frequency
NOS1 2
Cnr2 (Mus musculus)
Pain Link Frequency Relevance Heat
opioid receptor 8 100.00 Very High Very High Very High
Neuropathic pain 5 99.68 Very High Very High Very High
agonist 2 98.50 Very High Very High Very High
Antihyperalgesic 2 75.00 Quite High
antiallodynic 2 75.00 Quite High
Cannabinoid 2 75.00 Quite High
Opioid 1 75.00 Quite High
Spinal cord 1 69.20 Quite High
Enkephalin 1 49.16 Quite Low
Sciatic nerve 1 46.76 Quite Low
Disease Link Frequency Relevance Heat
Neuropathic Pain 5 99.68 Very High Very High Very High
Targeted Disruption 6 90.08 High High
Nervous System Injury 1 84.92 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Results suggest that inactivation of the nitric oxide-cGMP-PKG peripheral pathway triggered by NOS1 and NOS2 enhanced the peripheral actions of DOPr and CB2R agonists and that nitric oxide synthesized by NOS1 is implicated in the peripheral regulation of DOPr and CB2R gene transcription during neuropathic pain.
Regulation (regulation) of Transcription (transcription) of CB2R in NOS1 associated with neuropathic pain, agonist and opioid receptor
1) Confidence 0.37 Published 2010 Journal J. Pharmacol. Exp. Ther. Section Abstract Doc Link 20498253 Disease Relevance 0.82 Pain Relevance 0.74

General Comments

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