INT175260

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Context Info
Confidence 0.25
First Reported 2003
Last Reported 2007
Negated 0
Speculated 0
Reported most in Body
Documents 2
Total Number 2
Disease Relevance 4.41
Pain Relevance 0.21

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (SH2B2) signal transduction (SH2B2) plasma membrane (SH2B2)
cytoplasm (SH2B2) signal transducer activity (SH2B2)
Anatomy Link Frequency
central nervous system 2
SH2B2 (Homo sapiens)
Pain Link Frequency Relevance Heat
aspirin 29 98.34 Very High Very High Very High
Central nervous system 7 92.92 High High
alcohol 1 76.00 Quite High
cva 93 50.88 Quite High
abdominal pain 2 41.60 Quite Low
Inflammation 7 34.96 Quite Low
Multiple sclerosis 19 5.00 Very Low Very Low Very Low
imagery 14 5.00 Very Low Very Low Very Low
headache 10 5.00 Very Low Very Low Very Low
Migraine 7 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Anti-phospholipid Antibody Syndrome 142 100.00 Very High Very High Very High
Syndrome 53 100.00 Very High Very High Very High
Hepatolenticular Degeneration 1 99.32 Very High Very High Very High
Disease 40 98.70 Very High Very High Very High
Thrombophilia 155 98.00 Very High Very High Very High
Infection 13 97.74 Very High Very High Very High
Hepatitis C Virus Infection 5 97.54 Very High Very High Very High
Thrombosis Related Under Development 97 96.92 Very High Very High Very High
Pre-eclampsia 118 96.52 Very High Very High Very High
Thrombocytopenia 6 96.04 Very High Very High Very High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
At the same time it was found that 75% of LA patients were positive for HCV, which further supports the possible association between chronic HCV infection and aPLs85 Recently, a case of Wilson's disease (hepatolenticular degeneration) was reported along with primary APS in a patient presenting mainly with extrapyramidal damage.86 The latter was supposed to have had an autoimmune aPL component provoking mainly recurrent arterial/venous thrombosis and thrombocytopenia, but a possible systemic cellular involvement by aPLs in the central nervous system (besides thrombosis) may have interacted with a specific genetic background.51

Digestive manifestations

Negative_regulation (supposed) of Gene_expression (presenting) of APS in central nervous system associated with anti-phospholipid antibody syndrome, hepatolenticular degeneration, thrombosis related under development, disease, hepatitis c virus infection, infection, central nervous system and thrombocytopenia
1) Confidence 0.25 Published 2007 Journal Yonsei Medical Journal Section Body Doc Link PMC2628175 Disease Relevance 2.60 Pain Relevance 0.11
The combination of aspirin and heparin or low molecular weight (LMW) heparin is effective in recurrent fetal loss in APS syndrome and could be considered for women with inherited thrombophilias and history of severe preeclampsia, IUGR, abruptio placentae or fetal loss, although no controlled studies on the subject are currently available.
Negative_regulation (loss) of Gene_expression (syndrome) of APS associated with aspirin, thrombophilia, syndrome, abruptio placentae, anti-phospholipid antibody syndrome and pre-eclampsia
2) Confidence 0.03 Published 2003 Journal Reprod Biol Endocrinol Section Body Doc Link PMC305329 Disease Relevance 1.80 Pain Relevance 0.10

General Comments

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