INT181801

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Context Info
Confidence 0.29
First Reported 2005
Last Reported 2010
Negated 0
Speculated 1
Reported most in Body
Documents 3
Total Number 3
Disease Relevance 1.08
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

nucleoplasm (Atxn1) RNA binding (Atxn1) nucleus (Atxn1)
cytoplasm (Atxn1)
Atxn1 (Mus musculus)
Pain Link Frequency Relevance Heat
Calcium channel 2 22.64 Low Low
agonist 2 16.00 Low Low
Action potential 14 5.00 Very Low Very Low Very Low
cerebral cortex 6 5.00 Very Low Very Low Very Low
imagery 4 5.00 Very Low Very Low Very Low
anesthesia 3 5.00 Very Low Very Low Very Low
Thoracotomy 2 5.00 Very Low Very Low Very Low
alcohol 2 5.00 Very Low Very Low Very Low
Hippocampus 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Repression 2 96.88 Very High Very High Very High
Disease 50 95.44 Very High Very High Very High
Myocardial Infarction 30 89.12 High High
Apoptosis 6 76.48 Quite High
Obesity 2 61.84 Quite High
Targeted Disruption 35 56.28 Quite High
Aging 6 54.44 Quite High
Dilated Cardiomyopathy 2 53.48 Quite High
Cerebellar Ataxia 1 41.76 Quite Low
Werner Syndrome 1 36.68 Quite Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Meanwhile, DNA damage by itself is shared by the SCA1 pathology through high mobility group protein B proteins (Qi et al., 2007), and it is still possible that other molecules regulate DNA damage in SCA1 in addition to HMGBs.
Spec (possible) Regulation (regulate) of SCA1
1) Confidence 0.29 Published 2010 Journal The Journal of Cell Biology Section Body Doc Link PMC2867301 Disease Relevance 0.77 Pain Relevance 0
population of CPS cells is replated onto a nontagged adherent monolayer of the purified Sca-1+ cells, the fluorescent Sca-1?
Regulation (replated) of Sca-1
2) Confidence 0.23 Published 2005 Journal PLoS Biology Section Body Doc Link PMC1064849 Disease Relevance 0.06 Pain Relevance 0
Thus, there is likely a role for the Sca-1+ fraction in engraftment efficiency through any number of mechanisms, including migration, tropism, and entrapment.
Regulation (role) of Sca-1
3) Confidence 0.23 Published 2005 Journal PLoS Biology Section Body Doc Link PMC1064849 Disease Relevance 0.25 Pain Relevance 0

General Comments

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