INT182545

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Context Info
Confidence 0.16
First Reported 2005
Last Reported 2005
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.08
Pain Relevance 0.42

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

signal transduction (Ptgir) plasma membrane (Ptgir) signal transducer activity (Ptgir)
Ptgir (Mus musculus)
Pain Link Frequency Relevance Heat
qutenza 50 98.84 Very High Very High Very High
dorsal root ganglion 27 83.00 Quite High
antagonist 3 68.92 Quite High
agonist 2 57.88 Quite High
Inflammation 18 16.76 Low Low
Thermal hyperalgesia 15 11.80 Low Low
Pain 7 5.00 Very Low Very Low Very Low
withdrawal 5 5.00 Very Low Very Low Very Low
IPN 4 5.00 Very Low Very Low Very Low
Inflammatory mediators 3 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Ganglion Cysts 27 83.00 Quite High
Nociception 11 50.00 Quite Low
INFLAMMATION 23 16.76 Low Low
Hyperalgesia 18 11.80 Low Low
Pain 8 5.00 Very Low Very Low Very Low
Inflammatory Pain 4 5.00 Very Low Very Low Very Low
Sprains And Strains 3 5.00 Very Low Very Low Very Low
Injury 2 5.00 Very Low Very Low Very Low
Hypersensitivity 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
This property might explain the dose-dependent effects of PGI2 on capsaicin-activated currents: PKC-dependent sensitization of TRPV1 occurs downstream of Gq-coupled IP receptor activation at high concentrations (1000 nM) of PGI2 (1.5 min) while long (6.5 min) treatment with low concentrations (100 nM) of PGI2 causes potentiation of TRPV1 activity through Gs activation.
Positive_regulation (occurs) of Positive_regulation (activation) of IP associated with qutenza
1) Confidence 0.16 Published 2005 Journal Mol Pain Section Body Doc Link PMC1074353 Disease Relevance 0.08 Pain Relevance 0.42

General Comments

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