INT182771

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Context Info
Confidence 0.00
First Reported 2005
Last Reported 2005
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 2
Disease Relevance 3.83
Pain Relevance 0.28

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

nucleoplasm (NFKB1) cell differentiation (ROS1) mitochondrion (NFKB1)
cytoplasm (NFKB1) cell proliferation (ROS1) cytosol (NFKB1)
ROS1 (Homo sapiens)
NFKB1 (Homo sapiens)
CRP (Homo sapiens)
Pain Link Frequency Relevance Heat
Inflammatory marker 2 100.00 Very High Very High Very High
Inflammatory mediators 2 100.00 Very High Very High Very High
Osteoarthritis 4 76.44 Quite High
imagery 4 38.04 Quite Low
Inflammation 30 5.00 Very Low Very Low Very Low
Pain 16 5.00 Very Low Very Low Very Low
cytokine 8 5.00 Very Low Very Low Very Low
Tetrahydrobiopterin 6 5.00 Very Low Very Low Very Low
ischemia 4 5.00 Very Low Very Low Very Low
fibrosis 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
INFLAMMATION 34 100.00 Very High Very High Very High
Calcification 196 99.76 Very High Very High Very High
Atherosclerosis 78 98.70 Very High Very High Very High
Diabetes Mellitus 106 98.38 Very High Very High Very High
Atherosclerotic Plaque 6 89.44 High High
Metabolic Syndrome 14 87.28 High High
Osteoporosis 14 80.56 Quite High
Injury 16 78.12 Quite High
Osteoarthritis 4 76.44 Quite High
Chronic Renal Failure 90 50.00 Quite Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
In atheroscleropathy and non-diabetic atherosclerosis, the multiple metabolic toxicities and the resultant – parallel ROS, via stimulation of the essential and omnipotent nuclear factor (NFkappaB), which activates the downstream inflammatory mediators TNFalpha, IL-1, IL-6, and other inflammatory markers such as C-reactive protein (CRP).
ROS Positive_regulation (stimulation) of NFkappaB Positive_regulation (activates) of C-reactive protein associated with inflammatory mediators, atherosclerosis, diabetes mellitus and inflammatory marker
1) Confidence 0.00 Published 2005 Journal Cardiovasc Diabetol Section Body Doc Link PMC1079905 Disease Relevance 1.92 Pain Relevance 0.14
In atheroscleropathy and non-diabetic atherosclerosis, the multiple metabolic toxicities and the resultant – parallel ROS, via stimulation of the essential and omnipotent nuclear factor (NFkappaB), which activates the downstream inflammatory mediators TNFalpha, IL-1, IL-6, and other inflammatory markers such as C-reactive protein (CRP).
ROS Positive_regulation (stimulation) of NFkappaB Positive_regulation (activates) of CRP associated with inflammatory mediators, atherosclerosis, diabetes mellitus and inflammatory marker
2) Confidence 0.00 Published 2005 Journal Cardiovasc Diabetol Section Body Doc Link PMC1079905 Disease Relevance 1.92 Pain Relevance 0.14

General Comments

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