INT201755

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Context Info
Confidence 0.00
First Reported 2006
Last Reported 2006
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 1.40
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cell differentiation (ROS1) mitochondrion (Casp3) intracellular (Casp3)
cytoplasm (Casp3) cell proliferation (ROS1) cytosol (Casp3)
ROS1 (Homo sapiens)
Casp3 (Mus musculus)
Pain Link Frequency Relevance Heat
Kinase C 5 46.32 Quite Low
Eae 6 7.68 Low Low
Inflammation 9 5.00 Very Low Very Low Very Low
cytokine 8 5.00 Very Low Very Low Very Low
Hippocampus 7 5.00 Very Low Very Low Very Low
long-term potentiation 5 5.00 Very Low Very Low Very Low
Neurotransmitter 5 5.00 Very Low Very Low Very Low
Central nervous system 4 5.00 Very Low Very Low Very Low
Neuropeptide 2 5.00 Very Low Very Low Very Low
Inflammatory mediators 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Disease 127 97.16 Very High Very High Very High
Neurodegenerative Disease 36 96.92 Very High Very High Very High
Aging 5 93.72 High High
Stress 32 92.16 High High
Toxicity 15 88.72 High High
Drug Induced Neurotoxicity 4 84.24 Quite High
Death 56 80.88 Quite High
Amyloid Plaque 5 78.44 Quite High
Apoptosis 133 75.76 Quite High
Injury 7 61.84 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Moreover, ROS-mediated activation of caspase-3, can further stimulate sphingomyelinase and ceramide production, thus generating a feed-forward loop.
ROS Positive_regulation (activation) of caspase-3
1) Confidence 0.00 Published 2006 Journal BMC Neurosci Section Body Doc Link PMC1775042 Disease Relevance 1.40 Pain Relevance 0

General Comments

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