INT201868

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Context Info
Confidence 0.00
First Reported 2006
Last Reported 2006
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.12
Pain Relevance 0.21

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

nucleus (PASK, POU5F2) lipid binding (PASK) Golgi apparatus (PASK)
cytoplasm (PASK) signal transducer activity (PASK)
Anatomy Link Frequency
blood 1
PASK (Homo sapiens)
POU5F2 (Homo sapiens)
Pain Link Frequency Relevance Heat
antagonist 28 100.00 Very High Very High Very High
agonist 16 92.92 High High
endometriosis 3 5.00 Very Low Very Low Very Low
Pain 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Hemorrhage 8 61.96 Quite High
Reprotox - General 1 11 58.72 Quite High
Congenital Anomalies 1 27.88 Quite Low
Uterine Fibroids 21 8.88 Low Low
Endometriosis (extended) 9 5.00 Very Low Very Low Very Low
Hyperplasia 4 5.00 Very Low Very Low Very Low
Apoptosis 4 5.00 Very Low Very Low Very Low
Uterine Hemorrhage 2 5.00 Very Low Very Low Very Low
Gynecological Disease 1 5.00 Very Low Very Low Very Low
Pain 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Based on experiments conducted in cynomolgus monkeys, we previously proposed that the endometrial antiproliferative effect of SPRM and PAs might be due to the reduction in endometrial blood flow as consequence of vascular changes in the endometrium [9].
PAs Regulation (effect) of SPRM in blood associated with antagonist
1) Confidence 0.00 Published 2006 Journal Reprod Biol Endocrinol Section Body Doc Link PMC1775068 Disease Relevance 0.12 Pain Relevance 0.21

General Comments

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