INT209633

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Context Info
Confidence 0.06
First Reported 2007
Last Reported 2007
Negated 0
Speculated 1
Reported most in Body
Documents 1
Total Number 2
Disease Relevance 1.19
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

nucleoplasm (AR) transport (AR) enzyme binding (AR)
DNA binding (AR) cell-cell signaling (AR) cytoplasm (AR)
AR (Homo sapiens)
PCa (Homo sapiens)
Pain Link Frequency Relevance Heat
Kinase C 6 5.00 Very Low Very Low Very Low
addiction 2 5.00 Very Low Very Low Very Low
Vanilloid receptor-like 3 2 5.00 Very Low Very Low Very Low
Glutamate 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Prostate Cancer 166 100.00 Very High Very High Very High
Cancer 50 97.20 Very High Very High Very High
Disease 14 93.96 High High
Metastasis 10 78.72 Quite High
Disease Progression 4 65.28 Quite High
Repression 14 39.28 Quite Low
Apoptosis 10 5.00 Very Low Very Low Very Low
Williams-beuren Syndrome 4 5.00 Very Low Very Low Very Low
Genetic Translocation 2 5.00 Very Low Very Low Very Low
Stress 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Evidence from PCa biopsies for in vivo AR-mediated regulation of genes disclosed by ChIP Display
AR Regulation (regulation) of Gene_expression (biopsies) of PCa associated with prostate cancer
1) Confidence 0.06 Published 2007 Journal Mol Cancer Section Body Doc Link PMC1904239 Disease Relevance 0.36 Pain Relevance 0
To examine whether genes found in proximity to AR-occupied regions in our culture model are potentially regulated by the AR during PCa progression, we mined our microarray database of gene expression profiles in PCa tumors [24].
AR Spec (whether) Regulation (regulated) of Gene_expression (progression) of PCa associated with cancer and prostate cancer
2) Confidence 0.06 Published 2007 Journal Mol Cancer Section Body Doc Link PMC1904239 Disease Relevance 0.83 Pain Relevance 0

General Comments

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