INT21310

From wiki-pain
Revision as of 18:15, 20 September 2012 by Daniel (Talk | contribs)

(diff) ← Older revision | Latest revision (diff) | Newer revision → (diff)
Jump to: navigation, search
Context Info
Confidence 0.00
First Reported 1990
Last Reported 1990
Negated 0
Speculated 1
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0
Pain Relevance 0.09

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

extracellular region (Gnrh1, CGA) signal transduction (CGA) extracellular space (Gnrh1)
small molecule metabolic process (CGA) molecular_function (Gnrh1) cellular nitrogen compound metabolic process (CGA)
Anatomy Link Frequency
pituitary 3
Gnrh1 (Rattus norvegicus)
CGA (Homo sapiens)
Pain Link Frequency Relevance Heat
conotoxin 2 75.00 Quite High
tetrodotoxin 1 55.04 Quite High

|}

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
We have examined the pharmacology of the voltage-sensitive Ca2+ channels (VSCCs) that mediate gonadotropin secretion from primary cultures of rat pituitary cells, stimulated by either cell depolarization or by binding of gonadotropin-releasing hormone (GnRH).
GnRH Binding (binding) of in pituitary Spec (examined) Positive_regulation (mediate) of Localization (secretion) of gonadotropin in pituitary
1) Confidence 0.00 Published 1990 Journal Mol. Cell. Endocrinol. Section Abstract Doc Link 2170211 Disease Relevance 0 Pain Relevance 0.09

General Comments

This test has worked.

Personal tools
Namespaces

Variants
Actions
Navigation
Toolbox