INT222552

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Context Info
Confidence 0.00
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0
Pain Relevance 0.67

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

mitochondrion (Abat, Gh1) cytosol (Gh1) signal transduction (Gh1)
extracellular space (Gh1) extracellular region (Gh1) plasma membrane (Gh1)
Anatomy Link Frequency
pituitary 2
Abat (Rattus norvegicus)
Gh1 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
gABA 15 100.00 Very High Very High Very High
Glutamate 12 85.12 High High
antagonist 39 78.88 Quite High
agonist 22 63.60 Quite High
Glutamate receptor 1 58.28 Quite High
Neurotransmitter 8 48.04 Quite Low
Peripheral nervous system 1 25.36 Quite Low
Nerve growth factor 28 5.00 Very Low Very Low Very Low
Inflammation 27 5.00 Very Low Very Low Very Low
cytokine 22 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Targeted Disruption 22 29.44 Quite Low
Stress 48 7.68 Low Low
INFLAMMATION 28 5.00 Very Low Very Low Very Low
Cancer 22 5.00 Very Low Very Low Very Low
Pituitary Cancer 11 5.00 Very Low Very Low Very Low
Starvation 10 5.00 Very Low Very Low Very Low
Rheumatoid Arthritis 8 5.00 Very Low Very Low Very Low
Natriuresis 7 5.00 Very Low Very Low Very Low
Adhesions 6 5.00 Very Low Very Low Very Low
Hyperplasia 6 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
GABA stimulates GH production via a GABAB receptor, whereas blocking GABAB receptors with phaclofen decreased GH levels in pituitary cell cultures, demonstrating endogenous autocrine or paracrine GABA-ergic modulation of GH production (770).
GABA-ergic Regulation (modulation) of Gene_expression (production) of GH in pituitary associated with gaba
1) Confidence 0.00 Published 2008 Journal Journal of Neuroendocrinology Section Body Doc Link PMC2229370 Disease Relevance 0 Pain Relevance 0.67

General Comments

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