INT234307

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Context Info
Confidence 0.14
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.16
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

transport (Gria4, Gopc) plasma membrane (Gria4, Gopc) Golgi apparatus (Gopc)
cytoplasm (Gopc)
Gria4 (Mus musculus)
Gopc (Mus musculus)
Pain Link Frequency Relevance Heat
Thalamus 13 9.92 Low Low
cerebral cortex 4 5.00 Very Low Very Low Very Low
Glutamate receptor 4 5.00 Very Low Very Low Very Low
Action potential 4 5.00 Very Low Very Low Very Low
midbrain 2 5.00 Very Low Very Low Very Low
Glutamate 2 5.00 Very Low Very Low Very Low
tetrodotoxin 1 5.00 Very Low Very Low Very Low
Analgesic 1 5.00 Very Low Very Low Very Low
GABA receptor 1 5.00 Very Low Very Low Very Low
sodium channel 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Absence Epilepsy 15 81.88 Quite High
Ataxia 5 78.16 Quite High
Targeted Disruption 9 50.00 Quite Low
Sprains And Strains 20 5.00 Very Low Very Low Very Low
Convulsion 5 5.00 Very Low Very Low Very Low
Epilepsy 2 5.00 Very Low Very Low Very Low
Congenital Anomalies 2 5.00 Very Low Very Low Very Low
Generalized Epilepsy 2 5.00 Very Low Very Low Very Low
Death 1 5.00 Very Low Very Low Very Low
Dislocations 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
However, double mutant mice show a much lower incidence of SWD than do Gria4spkw1 homozygotes alone (Fig. 1; Gria3 ko, spwk1 double mutants); this result, observed in the first backcross, or N2 generation, was also confirmed in the N4 generation (Fig. 1) supportive of the primacy of the Gria3–Gria4 genetic interaction.
Gria4 Binding (interaction) of Fig
1) Confidence 0.14 Published 2008 Journal Human Molecular Genetics Section Body Doc Link PMC2405903 Disease Relevance 0.16 Pain Relevance 0

General Comments

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