INT235589

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Context Info
Confidence 0.41
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 9
Disease Relevance 4.84
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

intracellular (ITSN2) cytoplasm (ITSN2)
Anatomy Link Frequency
neuronal 1
eye 1
ITSN2 (Homo sapiens)
Pain Link Frequency Relevance Heat
isoflurane 18 5.00 Very Low Very Low Very Low
anesthesia 9 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Sleep Disorders 783 100.00 Very High Very High Very High
Muscle Hypotonia 18 90.64 High High
Stress 18 5.00 Very Low Very Low Very Low
Sleep Initiation And Maintenance Disorders 9 5.00 Very Low Very Low Very Low
Death 9 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
D: drowsiness; EEG: electroencephalographic; FFT: fast Fourier transform; NREM sleep: non-rapid eye movement sleep; REM sleep: rapid eye movement sleep; SD: sleep deprivation; SWA: slow wave activity; SWS: slow wave sleep; W: wakefulness.


Negative_regulation (deprivation) of SWA in eye associated with sleep disorders
1) Confidence 0.41 Published 2008 Journal BMC Neurosci Section Body Doc Link PMC2424059 Disease Relevance 0.35 Pain Relevance 0
We also observed a marked decline in SWA over the course of the night, with EEG power in the 0.5–4 Hz range being highest in the first 2-h interval after sleep onset and declining steadily afterwards.
Negative_regulation (decline) of SWA
2) Confidence 0.41 Published 2008 Journal BMC Neurosci Section Body Doc Link PMC2424059 Disease Relevance 0.28 Pain Relevance 0
SWA during SWS was also higher, during the first 4 hours after sleep deprivation, relative to SWA at the same circadian time in baseline.
Negative_regulation (deprivation) of SWA associated with sleep disorders
3) Confidence 0.30 Published 2008 Journal BMC Neurosci Section Body Doc Link PMC2424059 Disease Relevance 0.97 Pain Relevance 0
In mammals, NREM sleep SWA is high at sleep onset, when sleep pressure is high, and decreases progressively to reach low levels in late sleep.
Negative_regulation (decreases) of SWA
4) Confidence 0.30 Published 2008 Journal BMC Neurosci Section Abstract Doc Link PMC2424059 Disease Relevance 0.09 Pain Relevance 0
SWA is highest at the beginning of the major sleep period and declines thereafter.


Negative_regulation (declines) of SWA
5) Confidence 0.30 Published 2008 Journal BMC Neurosci Section Body Doc Link PMC2424059 Disease Relevance 0.88 Pain Relevance 0
This raises the possibility that the pigeon was able to obtain significant amounts of SWA during the 24-hour deprivation, either through drowsiness or through local sleep within select neuronal populations.
Negative_regulation (deprivation) of SWA in neuronal associated with sleep disorders
6) Confidence 0.30 Published 2008 Journal BMC Neurosci Section Body Doc Link PMC2424059 Disease Relevance 0.61 Pain Relevance 0
During baseline, SWA during D, SWS, and NREM sleep (defined as D and SWS combined) was highest at the beginning of the major sleep period and declined thereafter.
Negative_regulation (declined) of SWA
7) Confidence 0.30 Published 2008 Journal BMC Neurosci Section Abstract Doc Link PMC2424059 Disease Relevance 0.35 Pain Relevance 0
A similar trend of declining SWA was also evident for drowsiness, and when SWA was calculated for all of NREM sleep (Fig. 3C, right panel).
Negative_regulation (declining) of SWA associated with sleep disorders
8) Confidence 0.30 Published 2008 Journal BMC Neurosci Section Body Doc Link PMC2424059 Disease Relevance 0.71 Pain Relevance 0
Based on these data, they concluded that pigeons do not show a mammalian-like compensatory rebound in SWA following sleep deprivation.
Negative_regulation (deprivation) of SWA associated with sleep disorders
9) Confidence 0.30 Published 2008 Journal BMC Neurosci Section Body Doc Link PMC2424059 Disease Relevance 0.60 Pain Relevance 0

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