INT244454

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Context Info
Confidence 0.05
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.41
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

endoplasmic reticulum (EIF2AK3, SIL1) unfolded protein binding (SIL1) protein folding (SIL1)
cell (EIF2AK3) response to stress (EIF2AK3) cytoplasm (EIF2AK3)
EIF2AK3 (Homo sapiens)
SIL1 (Homo sapiens)
Pain Link Frequency Relevance Heat
Nicotine 9 5.00 Very Low Very Low Very Low
Inflammation 5 5.00 Very Low Very Low Very Low
addiction 1 5.00 Very Low Very Low Very Low
COX-2 inhibitor 1 5.00 Very Low Very Low Very Low
cINOD 1 5.00 Very Low Very Low Very Low
Inflammatory response 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Stress 67 99.22 Very High Very High Very High
Cancer 40 89.20 High High
Disease 7 88.52 High High
Malignant Neoplastic Disease 20 53.12 Quite High
Lung Cancer 47 5.00 Very Low Very Low Very Low
Nicotine Addiction 30 5.00 Very Low Very Low Very Low
Hypoxia 12 5.00 Very Low Very Low Very Low
Carcinoma 12 5.00 Very Low Very Low Very Low
Non-small-cell Lung Cancer 9 5.00 Very Low Very Low Very Low
Apoptosis 8 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The canonical pathway for ER stress activation of the UPR begins with the dissociation of BiP from PERK, resulting in PERK autophosphorylation.
PERK Binding (dissociation) of BiP associated with stress
1) Confidence 0.05 Published 2008 Journal BMC Cancer Section Body Doc Link PMC2527015 Disease Relevance 0.41 Pain Relevance 0

General Comments

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