INT249271

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Context Info
Confidence 0.06
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.34
Pain Relevance 0.09

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (CISH) intracellular (CISH) molecular_function (CISH)
cellular_component (CISH)
CISH (Homo sapiens)
Pain Link Frequency Relevance Heat
Inflammation 8 92.92 High High
TRP channel 2 36.48 Quite Low
cINOD 1 21.64 Low Low
agonist 4 5.00 Very Low Very Low Very Low
addiction 3 5.00 Very Low Very Low Very Low
Inflammatory response 2 5.00 Very Low Very Low Very Low
antagonist 2 5.00 Very Low Very Low Very Low
cytokine 1 5.00 Very Low Very Low Very Low
fibrosis 1 5.00 Very Low Very Low Very Low
Inflammatory mediators 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
INFLAMMATION 11 92.92 High High
Disease 5 92.40 High High
Adhesions 5 5.00 Very Low Very Low Very Low
Targeted Disruption 3 5.00 Very Low Very Low Very Low
Pulmonary Disease 2 5.00 Very Low Very Low Very Low
Neutrophil Disorders 2 5.00 Very Low Very Low Very Low
Stress 2 5.00 Very Low Very Low Very Low
Respiratory Syncytial Virus 1 5.00 Very Low Very Low Very Low
Toxicity 1 5.00 Very Low Very Low Very Low
Prostate Cancer 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Dissection of the mechanism linking Ca2+ store depletion to SOCs channel activation as well as the nature of these Ca2+ channels might help to characterize the regulation of superoxide anion release and serve as targets for pharmacological drug development in inflammatory diseases.



Negative_regulation (depletion) of Positive_regulation (activation) of SOCs associated with inflammation and disease
1) Confidence 0.06 Published 2008 Journal J Leukoc Biol Section Body Doc Link PMC2567897 Disease Relevance 0.34 Pain Relevance 0.09

General Comments

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