INT252415

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Context Info
Confidence 0.08
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.21
Pain Relevance 0.16

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

plasma membrane (Entpd8)
Entpd8 (Mus musculus)
Pain Term Frequency Confidence Heat
adenocard 22 99.54 Very High Very High Very High
Glutamate 20 93.08 High High
long-term potentiation 2 47.36 Quite Low
depression 7 41.76 Quite Low
Migraine 1 5.00 Very Low Very Low Very Low
cerebral cortex 1 5.00 Very Low Very Low Very Low
Central nervous system 5 5.00 Very Low Very Low Very Low
Antiepileptic agent 1 5.00 Very Low Very Low Very Low
antiepileptic Drug 8 5.00 Very Low Very Low Very Low
nMDA receptor 3 5.00 Very Low Very Low Very Low
Disease Term Frequency Confidence Heat
Targeted Disruption 1 90.80 High High
Epilepsy 29 65.04 Quite High
Convulsion 11 51.60 Quite High
Depression 7 41.76 Quite Low
Brain Injury 1 9.52 Low Low
Thrombosis 1 5.00 Very Low Very Low Very Low
Poisoning 1 5.00 Very Low Very Low Very Low
Hypoxia 1 5.00 Very Low Very Low Very Low
Death 1 5.00 Very Low Very Low Very Low
Absence Epilepsy 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The mechanism for this is that ATP is rapidly broken down into adenosine by ectonucleotidases (E-NTPDase, E-NPP, alkaline phosphatises, and ecto-5?
Phosphorylation (phosphatises) of E-NTPDase associated with adenocard
1) Confidence 0.08 Published 2008 Journal Purinergic Signal Section Body Doc Link PMC2583203 Disease Relevance 0.21 Pain Relevance 0.16

General Comments

This test has worked.

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