INT257738

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Context Info
Confidence 0.16
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 1.03
Pain Relevance 0.39

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (PIK3CA) signal transduction (PIK3CA) extracellular region (ACTN4)
nucleus (ACTN4) protein complex (ACTN4) cytoplasm (ACTN4)
PIK3CA (Homo sapiens)
ACTN4 (Homo sapiens)
Pain Link Frequency Relevance Heat
endometriosis 108 99.16 Very High Very High Very High
pain pelvic 2 5.00 Very Low Very Low Very Low
cytokine 1 5.00 Very Low Very Low Very Low
Inflammation 1 5.00 Very Low Very Low Very Low
Thalamus 1 5.00 Very Low Very Low Very Low
Dismenorea 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Endometriosis 138 99.16 Very High Very High Very High
Ovarian Cancer 15 95.08 Very High Very High Very High
Disease 11 40.00 Quite Low
Endometroid Carcinoma 1 33.32 Quite Low
Cancer 3 24.64 Low Low
Carcinoma 3 13.60 Low Low
Reprotox - General 3 4 5.00 Very Low Very Low Very Low
Myelodysplastic Syndromes 2 5.00 Very Low Very Low Very Low
Malignant Neoplastic Disease 2 5.00 Very Low Very Low Very Low
Reprotox - General 1 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Furthermore, since PI3K can control nuclear translocation of ACTN4, activation of the PI3K-Akt pathway in endometriosis may facilitate enhanced cytoplasmic expression of ACTN4, and may contribute the cell motility and invasion observed in endometriosis.
PI3K Regulation (control) of Localization (translocation) of ACTN4 associated with endometriosis
1) Confidence 0.16 Published 2008 Journal Reprod Biol Endocrinol Section Body Doc Link PMC2615013 Disease Relevance 1.03 Pain Relevance 0.39

General Comments

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