INT275045

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Context Info
Confidence 0.15
First Reported 2007
Last Reported 2009
Negated 0
Speculated 0
Reported most in Body
Documents 2
Total Number 2
Disease Relevance 1.32
Pain Relevance 0.31

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cell differentiation (MDK) signal transduction (MDK) extracellular region (MDK)
MDK (Homo sapiens)
Pain Link Frequency Relevance Heat
Inflammation 4 95.36 Very High Very High Very High
dexamethasone 48 86.48 High High
corticosteroid 11 67.40 Quite High
Inflammatory response 2 5.00 Very Low Very Low Very Low
ketamine 1 5.00 Very Low Very Low Very Low
anesthesia 1 5.00 Very Low Very Low Very Low
Pain 1 5.00 Very Low Very Low Very Low
alcohol 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Rheumatoid Arthritis 46 99.16 Very High Very High Very High
Meningoencephalitis 1 98.72 Very High Very High Very High
Cancer 1 96.12 Very High Very High Very High
INFLAMMATION 5 95.36 Very High Very High Very High
Keratitis 51 91.84 High High
Infection 40 89.68 High High
Injury 7 60.64 Quite High
Diabetes Mellitus 1 54.96 Quite High
Disorders Of The Lacrimal System 1 54.20 Quite High
Hypoxia 2 52.08 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
MK, a 13 kDa heparin-binding growth factor, is a RA-responsive gene involved in numerous processes including cell migration, tumor progression, inflammation, and angiogenesis.
MK Binding (binding) of associated with inflammation, cancer and rheumatoid arthritis
1) Confidence 0.15 Published 2009 Journal Respir Res Section Body Doc Link PMC2739515 Disease Relevance 0.53 Pain Relevance 0.31
CL-MK has been associated with other pathogens including acanthamoeba spp. and, rarely fungi.
CL-MK Binding (associated) of associated with meningoencephalitis
2) Confidence 0.00 Published 2007 Journal Clinical ophthalmology (Auckland, N.Z.) Section Body Doc Link PMC2704532 Disease Relevance 0.79 Pain Relevance 0

General Comments

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