INT281987

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Context Info
Confidence 0.08
First Reported 2008
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 2
Total Number 3
Disease Relevance 1.22
Pain Relevance 0.10

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

extracellular region (Crisp1)
Anatomy Link Frequency
osteoblasts 1
posterior 1
Crisp1 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
Central grey 4 92.48 High High
anesthesia 5 51.12 Quite High
Pain 1 49.08 Quite Low
Pyramidal cell 8 36.24 Quite Low
dexamethasone 3 5.00 Very Low Very Low Very Low
cerebral cortex 2 5.00 Very Low Very Low Very Low
ketamine 2 5.00 Very Low Very Low Very Low
midbrain 2 5.00 Very Low Very Low Very Low
depression 2 5.00 Very Low Very Low Very Low
Buprenorphine 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Arterial-venous Cerebral Malformations 40 99.34 Very High Very High Very High
Osteogenic Sarcomas 1 96.04 Very High Very High Very High
Osteoporosis 2 95.32 Very High Very High Very High
Urological Neuroanatomy 8 92.68 High High
Scoliosis 1 92.28 High High
Apoptosis 12 57.92 Quite High
Obesity 3 55.20 Quite High
Peripheral Arterial Disease 1 23.28 Low Low
Cancer 2 14.92 Low Low
Osteomyelitis 1 13.76 Low Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
In these cells, low concentrations of melatonin increased the mRNA levels of several genes expressed in osteoblasts including bone sialoprotein (BSP), alkaline phosphatase (ALP), osteopontin and osteocalcin.[15] Several studies using various animal models show that melatonin prevents bone deterioration including preventing idiopathic scoliosis in adolescents[1316] and that it stimulates proliferation of normal cells such as human bone cells.[13] However, there are no reports of melatonin effects on ADSC osteogenic differentiation.
Gene_expression (expressed) of sialoprotein in osteoblasts associated with scoliosis
1) Confidence 0.08 Published 2008 Journal Indian Journal of Plastic Surgery : Official Publication of the Association of Plastic Surgeons of India Section Body Doc Link PMC2739564 Disease Relevance 0.49 Pain Relevance 0.05
We conservatively refer to injections centered in the AVF and PSF as being located in the AEG and PEG, respectively, because, as indicated in Table 1, the size of the injection sites varied and, in some cases (for example F0523 and F0504), they were not restricted to those fields.
Gene_expression (respectively) of AEG associated with arterial-venous cerebral malformations
2) Confidence 0.05 Published 2010 Journal Frontiers in Neuroanatomy Section Body Doc Link PMC2904598 Disease Relevance 0.24 Pain Relevance 0
Labeling was most prominent in the AEG, particularly in the AVF and non-auditory areas located more anterior to the AVF, but was also present on the posterior EG (PEG), especially in its most posterior and dorsal part, where PSF is located.
Gene_expression (prominent) of AEG in posterior associated with arterial-venous cerebral malformations
3) Confidence 0.05 Published 2010 Journal Frontiers in Neuroanatomy Section Body Doc Link PMC2904598 Disease Relevance 0.49 Pain Relevance 0.05

General Comments

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