INT283948

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Context Info
Confidence 0.00
First Reported 2009
Last Reported 2009
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.21
Pain Relevance 0.22

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

Golgi apparatus (Ptgfrn) endoplasmic reticulum (Ptgfrn) nucleus (SALL1)
intracellular (SALL1) DNA binding (SALL1) cytoplasm (SALL1)
SALL1 (Homo sapiens)
Ptgfrn (Rattus norvegicus)
Pain Link Frequency Relevance Heat
long-term potentiation 40 94.64 High High
Lasting pain 20 66.84 Quite High
imagery 9 44.84 Quite Low
Pain 41 5.00 Very Low Very Low Very Low
nMDA receptor 7 5.00 Very Low Very Low Very Low
Pyramidal cell 7 5.00 Very Low Very Low Very Low
Glutamate receptor 5 5.00 Very Low Very Low Very Low
Central nervous system 4 5.00 Very Low Very Low Very Low
antagonist 3 5.00 Very Low Very Low Very Low
Inflammation 3 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Nociception 36 84.92 Quite High
Pain 59 66.84 Quite High
Cognitive Disorder 6 5.00 Very Low Very Low Very Low
Injury 4 5.00 Very Low Very Low Very Low
INFLAMMATION 3 5.00 Very Low Very Low Very Low
Inflammatory Pain 3 5.00 Very Low Very Low Very Low
Stress 2 5.00 Very Low Very Low Very Low
Anxiety Disorder 2 5.00 Very Low Very Low Very Low
Neuropathic Pain 2 5.00 Very Low Very Low Very Low
Hyperalgesia 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
It was apparent that TBS caused a significant increase in the amplitude of both positive- and negative-going fEPSP in saline control group of slices (Fig. 1B, C).
TBS Positive_regulation (caused) of Positive_regulation (increase) of negative-going fEPSP
1) Confidence 0.00 Published 2009 Journal Mol Pain Section Body Doc Link PMC2759921 Disease Relevance 0.21 Pain Relevance 0.22

General Comments

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