INT291545

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Context Info
Confidence 0.14
First Reported 2010
Last Reported 2010
Negated 0
Speculated 1
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0.44
Pain Relevance 0.37

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

mitochondrion (Dlat) Golgi apparatus (St8sia2) transferase activity, transferring acyl groups (Dlat)
Dlat (Rattus norvegicus)
St8sia2 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
agonist 13 100.00 Very High Very High Very High
ischemia 54 98.96 Very High Very High Very High
withdrawal 8 96.52 Very High Very High Very High
Pyramidal cell 22 65.68 Quite High
Hippocampus 9 5.00 Very Low Very Low Very Low
Neuronal excitability 5 5.00 Very Low Very Low Very Low
anesthesia 1 5.00 Very Low Very Low Very Low
nMDA receptor 1 5.00 Very Low Very Low Very Low
Glutamate 1 5.00 Very Low Very Low Very Low
antagonist 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Cv Unclassified Under Development 52 98.96 Very High Very High Very High
Aging 4 93.84 High High
Death 6 5.00 Very Low Very Low Very Low
Reprotox - General 1 4 5.00 Very Low Very Low Very Low
Middle Cerebral Artery Infarction 4 5.00 Very Low Very Low Very Low
Stroke 4 5.00 Very Low Very Low Very Low
Injury 3 5.00 Very Low Very Low Very Low
Thrombosis 2 5.00 Very Low Very Low Very Low
Myocardial Infarction 2 5.00 Very Low Very Low Very Low
Endometriosis (extended) 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
We examined the neuroprotective effects of E2 and two agonists that bind to non-classical estrogen receptors, G1 and STX, when administered after ischemia in middle-aged rats after prolonged ovarian hormone withdrawal.
E2 Spec (examined) Binding (bind) of STX associated with ischemia, agonist and withdrawal
1) Confidence 0.14 Published 2010 Journal PLoS ONE Section Abstract Doc Link PMC2799530 Disease Relevance 0.44 Pain Relevance 0.37

General Comments

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