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Context Info
Confidence 0.06
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 1.02
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (ABL1) protein modification process (ABL1) mitochondrion (ABL1)
nucleolus (ABL1) nucleus (ABL1) DNA binding (ABL1)
ABL1 (Homo sapiens)
Pain Link Frequency Relevance Heat
qutenza 1 23.20 Low Low
Disease Link Frequency Relevance Heat
Myeloid Leukemia 12 99.86 Very High Very High Very High
Congenital Anomalies 1 97.84 Very High Very High Very High
Philadelphia Chromosome 2 86.44 High High
Genetic Translocation 1 83.84 Quite High
Chronic Myeloid Leukemia 8 81.12 Quite High
Shock 7 78.48 Quite High
Apoptosis 8 70.60 Quite High
Disease 1 53.96 Quite High
Cancer 5 39.28 Quite Low
Stress 11 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Hence the standard care for treating CML is to target Bcr Abl tyrosine kinase by the pharmacological inhibitors Imatinib or more potent Dasatinib and Nilotinib [17].
Regulation (target) of Gene_expression (tyrosine kinase) of Abl associated with myeloid leukemia
1) Confidence 0.06 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2806839 Disease Relevance 1.02 Pain Relevance 0

General Comments

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