INT294637

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Context Info
Confidence 0.15
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.97
Pain Relevance 0.09

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytoplasm (Bcl2a1a, Bcl2) mitochondrion (Bcl2) cell morphogenesis (Bcl2)
endoplasmic reticulum (Bcl2) intracellular (Bcl2) cell proliferation (Bcl2)
Bcl2a1a (Mus musculus)
Bcl2 (Mus musculus)
Pain Link Frequency Relevance Heat
ischemia 18 67.68 Quite High
addiction 1 61.60 Quite High
Inflammatory response 2 53.48 Quite High
cytokine 4 5.00 Very Low Very Low Very Low
Inflammation 4 5.00 Very Low Very Low Very Low
isoflurane 4 5.00 Very Low Very Low Very Low
anesthesia 2 5.00 Very Low Very Low Very Low
Buprenorphine 2 5.00 Very Low Very Low Very Low
halothane 2 5.00 Very Low Very Low Very Low
ketamine 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Apoptosis 43 100.00 Very High Very High Very High
Injury 40 89.56 High High
Cv Unclassified Under Development 18 67.68 Quite High
INFLAMMATION 6 53.08 Quite High
Death 7 6.96 Low Low
Necrosis 4 5.00 Very Low Very Low Very Low
Adhesions 3 5.00 Very Low Very Low Very Low
Sepsis 3 5.00 Very Low Very Low Very Low
Myocardial Infarction 3 5.00 Very Low Very Low Very Low
Exsanguination 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
These results suggest that one mechanism by which exogenously administered rhBCL2 or rhBCL2A1 protects tissue against I/R-induced apoptosis is by augmenting increased expression of endogenous mouse BCL2.
rhBCL2A1 Positive_regulation (augmenting) of Gene_expression (expression) of BCL2 associated with apoptosis
1) Confidence 0.15 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2816997 Disease Relevance 0.97 Pain Relevance 0.09

General Comments

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