INT295429

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Context Info
Confidence 0.42
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.84
Pain Relevance 0.07

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

signal transduction (KIT) extracellular space (KIT) plasma membrane (KIT)
nucleus (KIT) cytoplasm (KIT)
KIT (Homo sapiens)
Pain Link Frequency Relevance Heat
abdominal pain 1 65.80 Quite High
imagery 2 5.00 Very Low Very Low Very Low
Bioavailability 2 5.00 Very Low Very Low Very Low
headache 1 5.00 Very Low Very Low Very Low
cva 1 5.00 Very Low Very Low Very Low
positron emission tomography 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Gastrointestinal Stromal Tumor 104 98.58 Very High Very High Very High
Disease 62 82.60 Quite High
Disease Progression 12 80.40 Quite High
Asthenia 1 66.32 Quite High
Abdominal Pain 1 65.80 Quite High
Diarrhoea 2 65.16 Quite High
Toxicity 2 60.56 Quite High
Hypertension 1 9.04 Low Low
Syndrome 2 8.48 Low Low
Fatigue 1 7.28 Low Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
In contrast, KIT signaling in primary imatinib-resistant GIST shows no evidence of inhibition to imatinib and is similar to that seen in untreated GIST, indicating that KIT primary resistance is associated with persistent KIT phosphorylation and activation of downstream pathways.37 The molecular mechanisms responsible for primary-imatinib resistance differ from those of secondary resistance.
Positive_regulation (activation) of Phosphorylation (phosphorylation) of KIT associated with gastrointestinal stromal tumor
1) Confidence 0.42 Published 2010 Journal Biologics : Targets & Therapy Section Body Doc Link PMC2819895 Disease Relevance 0.84 Pain Relevance 0.07

General Comments

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