INT296040

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Context Info
Confidence 0.07
First Reported 2009
Last Reported 2009
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.70
Pain Relevance 0.12

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

nucleoplasm (TH1L) extracellular space (IL17A) cell death (IL17A)
extracellular region (IL17A) nucleus (TH1L) cell-cell signaling (IL17A)
Anatomy Link Frequency
T-cells 1
IL17A (Homo sapiens)
TH1L (Homo sapiens)
Pain Link Frequency Relevance Heat
cytokine 36 99.92 Very High Very High Very High
agonist 1 74.48 Quite High
Inflammation 32 50.00 Quite Low
Crohn's disease 4 44.48 Quite Low
tolerance 13 5.00 Very Low Very Low Very Low
adenocard 3 5.00 Very Low Very Low Very Low
chemokine 3 5.00 Very Low Very Low Very Low
withdrawal 1 5.00 Very Low Very Low Very Low
Nerve growth factor 1 5.00 Very Low Very Low Very Low
Inflammatory response 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Wasting Syndrome 1 97.00 Very High Very High Very High
Inflammatory Bowel Disease 30 88.96 High High
Diarrhoea 1 71.24 Quite High
Colitis 8 68.44 Quite High
Autoimmune Disease 12 65.60 Quite High
Disease 22 59.52 Quite High
Rheumatoid Arthritis 2 56.40 Quite High
INFLAMMATION 30 50.00 Quite Low
Cancer 33 5.00 Very Low Very Low Very Low
Hepatocellular Cancer 8 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
IL-17A can also modulate the TH1 polarization in vitro in inhibitory sense, suppressing the induction of the transcriptional factor T-bet, the “master regulator” of TH1 differentiation of T-cells.
IL-17A Regulation (modulate) of TH1 in T-cells
1) Confidence 0.07 Published 2009 Journal Mediators of Inflammation Section Body Doc Link PMC2821644 Disease Relevance 0.70 Pain Relevance 0.12

General Comments

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