INT304001

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Context Info
Confidence 0.07
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.35
Pain Relevance 0.20

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

signal transduction (Lep) extracellular space (Lep) extracellular region (Lep)
plasma membrane (ICAM3) intracellular (Lep) lipid metabolic process (Lep)
Anatomy Link Frequency
eosinophils 1
Lep (Mus musculus)
ICAM3 (Homo sapiens)
Pain Link Frequency Relevance Heat
Inflammation 9 90.96 High High
cytokine 24 89.92 High High
chemokine 3 87.36 High High
Inflammatory stimuli 2 5.00 Very Low Very Low Very Low
withdrawal 1 5.00 Very Low Very Low Very Low
endometriosis 1 5.00 Very Low Very Low Very Low
Inflammatory response 1 5.00 Very Low Very Low Very Low
spinal inflammation 1 5.00 Very Low Very Low Very Low
antagonist 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Adhesions 2 100.00 Very High Very High Very High
Diabetes Mellitus 1 94.52 High High
INFLAMMATION 12 90.96 High High
Uremia 1 62.56 Quite High
Apoptosis 6 41.76 Quite Low
Obesity 22 5.36 Low Low
Stress 3 5.00 Very Low Very Low Very Low
Autoimmune Disease 3 5.00 Very Low Very Low Very Low
Metabolic Syndrome 2 5.00 Very Low Very Low Very Low
Atherosclerosis 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
On eosinophils, leptin could upregulate cell surface expression of adhesion molecules ICAM-1 and CD18 but suppress ICAM-3 and L-selectin.
leptin Negative_regulation (suppress) of ICAM-3 in eosinophils associated with adhesions
1) Confidence 0.07 Published 2010 Journal Mediators of Inflammation Section Body Doc Link PMC2846344 Disease Relevance 0.35 Pain Relevance 0.20

General Comments

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