INT304104

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Context Info
Confidence 0.14
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 2
Disease Relevance 2.60
Pain Relevance 0.19

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytoplasm (Il13, AHR) immune system process (AHR) cell morphogenesis (AHR)
DNA binding (AHR) response to stress (AHR) signal transducer activity (AHR)
Il13 (Mus musculus)
AHR (Homo sapiens)
Pain Link Frequency Relevance Heat
Inflammation 28 99.16 Very High Very High Very High
antagonist 2 88.88 High High
aspirin 16 50.00 Quite Low
cytokine 4 24.80 Low Low
cINOD 4 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Rhinitis 38 99.96 Very High Very High Very High
INFLAMMATION 30 99.16 Very High Very High Very High
Asthma 164 98.80 Very High Very High Very High
Adhesions 2 46.76 Quite Low
Occupational Lung Diseases 4 40.56 Quite Low
Eosinophilia 6 30.64 Quite Low
Hypersensitivity 8 22.80 Low Low
Nasal Polyps 4 7.44 Low Low
Chronic Sinusitis 8 6.64 Low Low
Eczema 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
In ethnic German populations, the IL-13 Arg110Gln variant has a significant association with high IgE levels.13 In addition, this allele was associated with the asthma phenotype in British and Japanese populations.14 Moreover, IL-13 is necessary and sufficient for the induction of AHR eosinophilic airway inflammation; in the absence of IL-13, this does not progress to AHR.
IL-13 Positive_regulation (necessary) of Positive_regulation (induction) of AHR associated with asthma, rhinitis and inflammation
1) Confidence 0.14 Published 2010 Journal Allergy, Asthma & Immunology Research Section Body Doc Link PMC2846737 Disease Relevance 1.30 Pain Relevance 0.09
In ethnic German populations, the IL-13 Arg110Gln variant has a significant association with high IgE levels.13 In addition, this allele was associated with the asthma phenotype in British and Japanese populations.14 Moreover, IL-13 is necessary and sufficient for the induction of AHR eosinophilic airway inflammation; in the absence of IL-13, this does not progress to AHR.
IL-13 Positive_regulation (sufficient) of Positive_regulation (induction) of AHR associated with asthma, rhinitis and inflammation
2) Confidence 0.14 Published 2010 Journal Allergy, Asthma & Immunology Research Section Body Doc Link PMC2846737 Disease Relevance 1.30 Pain Relevance 0.09

General Comments

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