INT325313

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Context Info
Confidence 0.20
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.48
Pain Relevance 0.23

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

extracellular region (IL23A, IL22) extracellular space (IL22) cell-cell signaling (IL22)
Anatomy Link Frequency
Th17 cells 2
IL23A (Homo sapiens)
IL22 (Homo sapiens)
Pain Link Frequency Relevance Heat
Inflammation 7 80.60 Quite High
psoriasis 61 71.36 Quite High
cytokine 5 64.40 Quite High
chemokine 1 37.44 Quite Low
Arthritis 21 13.88 Low Low
depression 1 6.68 Low Low
withdrawal 8 5.00 Very Low Very Low Very Low
methotrexate 5 5.00 Very Low Very Low Very Low
corticosteroid 4 5.00 Very Low Very Low Very Low
Onset of action 3 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Psoriasis 69 84.96 Quite High
INFLAMMATION 6 80.60 Quite High
Autoimmune Disease 1 52.80 Quite High
Arthritis 2 13.88 Low Low
Cancer 2 13.28 Low Low
Heart Disease 1 12.96 Low Low
Disease 13 12.64 Low Low
Affective Disorder 1 8.48 Low Low
Depression 1 6.68 Low Low
Seronegative Spondarthritis 20 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
In turn, IL-23 sustains Th17 proliferation, hence primarily increasing levels of IL-17 and IL-22 produced by these activated Th17 cells (It has been suggested that Th0 differentiation into Th17 is originally initiated by IL-6 and TGF-?
IL-23 Positive_regulation (increasing) of Gene_expression (produced) of IL-22 in Th17 cells
1) Confidence 0.20 Published 2010 Journal Core Evidence Section Body Doc Link PMC2915500 Disease Relevance 0.48 Pain Relevance 0.23

General Comments

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