INT329188

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Context Info
Confidence 0.29
First Reported 2010
Last Reported 2010
Negated 2
Speculated 0
Reported most in Body
Documents 2
Total Number 4
Disease Relevance 1.34
Pain Relevance 0.19

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cell death (FOSL2) nucleolus (FOSL2) nucleus (FOSL2)
FOSL2 (Homo sapiens)
Pain Link Frequency Relevance Heat
Inflammatory response 3 90.08 High High
Inflammation 29 81.80 Quite High
psoriasis 22 66.96 Quite High
Glutamate 1 19.84 Low Low
metalloproteinase 1 17.00 Low Low
cytokine 15 5.00 Very Low Very Low Very Low
chemokine 7 5.00 Very Low Very Low Very Low
alcohol 3 5.00 Very Low Very Low Very Low
abdominal pain 3 5.00 Very Low Very Low Very Low
methotrexate 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Apoptosis 409 95.92 Very High Very High Very High
Cancer 26 95.84 Very High Very High Very High
INFLAMMATION 31 90.08 High High
Dermatitis 31 81.44 Quite High
Disease 20 78.72 Quite High
Infection 78 67.20 Quite High
Psoriasis 40 66.96 Quite High
Contact Dermatitis 9 66.08 Quite High
Targeted Disruption 7 38.12 Quite Low
Wound Healing 1 36.32 Quite Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Thus, the increase in magnitude of JUN expression seen in WTSTS could lead to the induction of FOSL2 and LTBP3 seen in these cells.
Positive_regulation (induction) of FOSL2
1) Confidence 0.29 Published 2010 Journal BMC Genomics Section Body Doc Link PMC2996966 Disease Relevance 0.16 Pain Relevance 0
Interestingly, FOSL2 or FRA-2 was induced here but not in the U versus WT comparison.
Neg (not) Positive_regulation (induced) of FRA-2
2) Confidence 0.29 Published 2010 Journal BMC Genomics Section Body Doc Link PMC2996966 Disease Relevance 0.18 Pain Relevance 0
Interestingly, FOSL2 or FRA-2 was induced here but not in the U versus WT comparison.
Neg (not) Positive_regulation (induced) of FOSL2
3) Confidence 0.29 Published 2010 Journal BMC Genomics Section Body Doc Link PMC2996966 Disease Relevance 0.18 Pain Relevance 0
Ascorbic acid is an antiapoptotic antioxidant, which provides redox-dependent inhibition of JNK and activation of Fra-1 and Fra-2.
Positive_regulation (activation) of Fra-2
4) Confidence 0.10 Published 2010 Journal Mediators of Inflammation Section Body Doc Link PMC2929514 Disease Relevance 0.82 Pain Relevance 0.19

General Comments

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