INT33148

From wiki-pain
Revision as of 21:39, 20 September 2012 by Daniel (Talk | contribs)

(diff) ← Older revision | Latest revision (diff) | Newer revision → (diff)
Jump to: navigation, search
Context Info
Confidence 0.25
First Reported 1985
Last Reported 2010
Negated 0
Speculated 0
Reported most in Abstract
Documents 4
Total Number 4
Disease Relevance 2.95
Pain Relevance 1.68

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cell adhesion (FPR2) plasma membrane (FPR2) signal transducer activity (FPR2)
Anatomy Link Frequency
neutrophils 1
FPR2 (Homo sapiens)
Pain Link Frequency Relevance Heat
Inflammation 50 100.00 Very High Very High Very High
narcan 9 100.00 Very High Very High Very High
corticosteroid 31 99.98 Very High Very High Very High
agonist 4 99.82 Very High Very High Very High
Inflammatory response 7 92.24 High High
dexamethasone 21 72.56 Quite High
cINOD 2 56.64 Quite High
opiate 3 54.72 Quite High
antagonist 1 52.16 Quite High
Inflammatory mediators 2 51.88 Quite High
Disease Link Frequency Relevance Heat
INFLAMMATION 52 100.00 Very High Very High Very High
Asthma 101 99.24 Very High Very High Very High
Sepsis 1 84.40 Quite High
Htlv Types I And Ii 2 81.72 Quite High
Inflammatory Bowel Disease 1 76.60 Quite High
Pulmonary Disease 2 72.96 Quite High
Systemic Sclerosis 1 72.44 Quite High
Hypersensitivity 1 62.28 Quite High
Rhinitis 3 61.92 Quite High
Disease 2 61.52 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
In summary, we demonstrate impaired corticosteroid modulation of the pro-inflammatory lipid mediator LTB4 but not of the anti-inflammatory lipoxin, LXA4, in AMs of severe asthma.
Regulation (modulation) of LXA4 associated with asthma, corticosteroid and inflammation
1) Confidence 0.25 Published 2010 Journal Respir Res Section Body Doc Link PMC2894769 Disease Relevance 1.26 Pain Relevance 0.40
In agreement with in vitro results, ALX agonists, namely LXA4 and 15-epi-LXA4 and their stable analogs, regulate PMN during acute inflammation.
Regulation (regulate) of LXA4 associated with inflammation and agonist
2) Confidence 0.15 Published 2004 Journal Rheum. Dis. Clin. North Am. Section Abstract Doc Link 15061569 Disease Relevance 0.80 Pain Relevance 0.44
Using the superoxide dismutase inhibitable reduction of cytochrome c assay, we studied, the effect of (-) naloxone on N-formyl-methionyl-leucyl-phenylalanine (FMLP) stimulated superoxide (O2-) release from human neutrophils.
Regulation (effect) of N-formyl-methionyl-leucyl-phenylalanine in neutrophils associated with narcan
3) Confidence 0.15 Published 1985 Journal Life Sci. Section Abstract Doc Link 2995744 Disease Relevance 0.08 Pain Relevance 0.41
In agreement with in vitro results, ALX agonists, namely LXA4 and 15-epi-LXA4 and their stable analogs, regulate PMN during acute inflammation.
Regulation (regulate) of LXA4 associated with inflammation and agonist
4) Confidence 0.13 Published 2004 Journal Rheum. Dis. Clin. North Am. Section Abstract Doc Link 15061569 Disease Relevance 0.80 Pain Relevance 0.44

General Comments

This test has worked.

Personal tools
Namespaces

Variants
Actions
Navigation
Toolbox