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Context Info
Confidence 0.41
First Reported 1975
Last Reported 1999
Negated 1
Speculated 0
Reported most in Abstract
Documents 3
Total Number 3
Disease Relevance 1.39
Pain Relevance 0.60

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

peptidase activity (Ren) extracellular space (Ren) extracellular region (Ren)
intracellular (Ren) response to stress (Ren)
Anatomy Link Frequency
plasma 3
liver 2
blood 1
Ren (Rattus norvegicus)
Pain Link Frequency Relevance Heat
fluoxetine 5 93.76 High High
sSRI 1 93.28 High High
headache 1 85.68 High High
Serotonin 10 77.28 Quite High
adenocard 1 50.48 Quite High
bradykinin 1 50.04 Quite High
Clonidine 1 49.56 Quite Low
midbrain 1 25.00 Low Low
Disease Link Frequency Relevance Heat
Pressure And Volume Under Development 3 99.28 Very High Very High Very High
Anxiety Disorder 3 98.44 Very High Very High Very High
Appetite Loss 4 97.52 Very High Very High Very High
Adenoma 1 90.56 High High
Hamartoma 2 90.12 High High
Stress 2 88.48 High High
Hypotension 4 87.04 High High
Headache 1 85.68 High High
Sprains And Strains 2 83.52 Quite High
Hyperphagia 1 52.56 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
It did not affect baseline plasma adrenocorticotropic hormone levels and renin activity, but decreased plasma corticosterone levels.
Neg (not) Regulation (affect) of Gene_expression (levels) of renin in plasma
1) Confidence 0.41 Published 1999 Journal Neuroscience Section Abstract Doc Link 10392854 Disease Relevance 0.59 Pain Relevance 0.46
Effects of the K+ channel activators, RP 52891, cromakalim and diazoxide, on the plasma insulin level, plasma renin activity and blood pressure in rats.
Regulation (Effects) of in blood Gene_expression (level) of renin in plasma
2) Confidence 0.40 Published 1991 Journal J. Pharmacol. Exp. Ther. Section Title Doc Link 1906538 Disease Relevance 0.20 Pain Relevance 0.05
The morphological characteristics of the hamartomatous abnormality are described and evidences are put forward which would suggest that the liver lesion might have been the site of the abnormal renin production which was responsible for the systemic arterial hypertension.
Regulation (responsible) of Gene_expression (production) of renin in liver associated with pressure and volume under development
3) Confidence 0.27 Published 1975 Journal Virchows Arch A Pathol Anat Histol Section Abstract Doc Link 804756 Disease Relevance 0.60 Pain Relevance 0.09

General Comments

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